Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cells

Abstract Transplanted adipose stem cells (ASC) have a low survival rate in the body, and there are not many ASC that can be effectively used, which weakens their tissue repair function. Based on this status quo, a new type of copper‐based metal–organic network (Cu‐MON) was used to pretreat cells to...

Full description

Saved in:
Bibliographic Details
Main Authors: Ruixin Pang, Kaijing Liu, Biou Liu, Jing Yang
Format: Article
Language:English
Published: Wiley 2025-03-01
Series:FASEB BioAdvances
Subjects:
Online Access:https://doi.org/10.1096/fba.2024-00153
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849772953511657472
author Ruixin Pang
Kaijing Liu
Biou Liu
Jing Yang
author_facet Ruixin Pang
Kaijing Liu
Biou Liu
Jing Yang
author_sort Ruixin Pang
collection DOAJ
description Abstract Transplanted adipose stem cells (ASC) have a low survival rate in the body, and there are not many ASC that can be effectively used, which weakens their tissue repair function. Based on this status quo, a new type of copper‐based metal–organic network (Cu‐MON) was used to pretreat cells to regulate cell activity in order to improve the efficacy of cell therapy or reduce the number of cells used, thus reducing the cost of clinical treatment. Gene expression changes before and after Cu‐MON treatment of normal donor adipose stem cells (ND‐ASC) and type 2 diabetes mellitus adipose stem cells (T2DM‐ASC) were evaluated through RNA sequencing, KEGG and GO enrichment analysis. The results showed that Cu‐MON improved ASC cell quality by regulating immune response and promoting paracrine secretion. IL‐17 signaling pathway and IL‐6, CXCL8, and MMP‐9 were key pathways and necessary genes that affected the ability of stem cells. In addition, Cu‐MON also improved stem cell antiviral ability through Type I interferon signaling pathway. Our research showed that Cu‐MON had improved the cell quality of ASC by regulating immune response, promoting paracrine secretion, and improving antiviral capabilities. This approach to biomaterial pretreatment is fast, convenient, and relatively safe, and provides new strategies for improving the efficiency of cell therapies.
format Article
id doaj-art-1a4236fb0c064b1bb3fd3e15edd25d0d
institution DOAJ
issn 2573-9832
language English
publishDate 2025-03-01
publisher Wiley
record_format Article
series FASEB BioAdvances
spelling doaj-art-1a4236fb0c064b1bb3fd3e15edd25d0d2025-08-20T03:02:11ZengWileyFASEB BioAdvances2573-98322025-03-0173n/an/a10.1096/fba.2024-00153Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cellsRuixin Pang0Kaijing Liu1Biou Liu2Jing Yang3Fourth Clinical Medical College & Fourth Affiliated Hospital of Harbin Medical University Harbin ChinaState Key Laboratory of Advanced Medical Materials and Devices, Institute of Biomedical Engineering Chinese Academy of Medical Science & Peking Union Medical College Tianjin ChinaDepartment of General Surgery Forth Affiliate Hospital of Harbin Medical University Harbin ChinaState Key Laboratory of Advanced Medical Materials and Devices, Institute of Biomedical Engineering Chinese Academy of Medical Science & Peking Union Medical College Tianjin ChinaAbstract Transplanted adipose stem cells (ASC) have a low survival rate in the body, and there are not many ASC that can be effectively used, which weakens their tissue repair function. Based on this status quo, a new type of copper‐based metal–organic network (Cu‐MON) was used to pretreat cells to regulate cell activity in order to improve the efficacy of cell therapy or reduce the number of cells used, thus reducing the cost of clinical treatment. Gene expression changes before and after Cu‐MON treatment of normal donor adipose stem cells (ND‐ASC) and type 2 diabetes mellitus adipose stem cells (T2DM‐ASC) were evaluated through RNA sequencing, KEGG and GO enrichment analysis. The results showed that Cu‐MON improved ASC cell quality by regulating immune response and promoting paracrine secretion. IL‐17 signaling pathway and IL‐6, CXCL8, and MMP‐9 were key pathways and necessary genes that affected the ability of stem cells. In addition, Cu‐MON also improved stem cell antiviral ability through Type I interferon signaling pathway. Our research showed that Cu‐MON had improved the cell quality of ASC by regulating immune response, promoting paracrine secretion, and improving antiviral capabilities. This approach to biomaterial pretreatment is fast, convenient, and relatively safe, and provides new strategies for improving the efficiency of cell therapies.https://doi.org/10.1096/fba.2024-00153gene expressionRNA sequence analysisstem cellstranscriptometype 2 diabetes mellitus
spellingShingle Ruixin Pang
Kaijing Liu
Biou Liu
Jing Yang
Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cells
FASEB BioAdvances
gene expression
RNA sequence analysis
stem cells
transcriptome
type 2 diabetes mellitus
title Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cells
title_full Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cells
title_fullStr Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cells
title_full_unstemmed Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cells
title_short Enhancement of adipose stem cell quality via Cu‐MON: Transcriptome and bioinformatics analysis of normal and diabetic stem cells
title_sort enhancement of adipose stem cell quality via cu mon transcriptome and bioinformatics analysis of normal and diabetic stem cells
topic gene expression
RNA sequence analysis
stem cells
transcriptome
type 2 diabetes mellitus
url https://doi.org/10.1096/fba.2024-00153
work_keys_str_mv AT ruixinpang enhancementofadiposestemcellqualityviacumontranscriptomeandbioinformaticsanalysisofnormalanddiabeticstemcells
AT kaijingliu enhancementofadiposestemcellqualityviacumontranscriptomeandbioinformaticsanalysisofnormalanddiabeticstemcells
AT biouliu enhancementofadiposestemcellqualityviacumontranscriptomeandbioinformaticsanalysisofnormalanddiabeticstemcells
AT jingyang enhancementofadiposestemcellqualityviacumontranscriptomeandbioinformaticsanalysisofnormalanddiabeticstemcells