Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transfer

In recent years, mitochondrial transfer has emerged as a universal phenomenon intertwined with various systemic physiological and pathological processes. Alzheimer’s disease (AD) is a multifactorial disease, with mitochondrial dysfunction at its core. Although numerous studies have found evidence of...

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Main Authors: Yun Wei, Xinlei Du, Hongling Guo, Jingjing Han, Meixia Liu
Format: Article
Language:English
Published: Frontiers Media S.A. 2024-12-01
Series:Frontiers in Aging Neuroscience
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fnagi.2024.1517965/full
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author Yun Wei
Xinlei Du
Hongling Guo
Jingjing Han
Meixia Liu
author_facet Yun Wei
Xinlei Du
Hongling Guo
Jingjing Han
Meixia Liu
author_sort Yun Wei
collection DOAJ
description In recent years, mitochondrial transfer has emerged as a universal phenomenon intertwined with various systemic physiological and pathological processes. Alzheimer’s disease (AD) is a multifactorial disease, with mitochondrial dysfunction at its core. Although numerous studies have found evidence of mitochondrial transfer in AD models, the precise mechanisms remain unclear. Recent studies have revealed the dynamic transfer of mitochondria in Alzheimer’s disease, not only between nerve cells and glial cells, but also between nerve cells and glial cells. In this review, we explore the pathways and mechanisms of mitochondrial transfer in Alzheimer’s disease and how these transfer activities contribute to disease progression.
format Article
id doaj-art-1a22b841021f4e8088a1eab887060a67
institution Kabale University
issn 1663-4365
language English
publishDate 2024-12-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Aging Neuroscience
spelling doaj-art-1a22b841021f4e8088a1eab887060a672024-12-17T06:23:20ZengFrontiers Media S.A.Frontiers in Aging Neuroscience1663-43652024-12-011610.3389/fnagi.2024.15179651517965Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transferYun WeiXinlei DuHongling GuoJingjing HanMeixia LiuIn recent years, mitochondrial transfer has emerged as a universal phenomenon intertwined with various systemic physiological and pathological processes. Alzheimer’s disease (AD) is a multifactorial disease, with mitochondrial dysfunction at its core. Although numerous studies have found evidence of mitochondrial transfer in AD models, the precise mechanisms remain unclear. Recent studies have revealed the dynamic transfer of mitochondria in Alzheimer’s disease, not only between nerve cells and glial cells, but also between nerve cells and glial cells. In this review, we explore the pathways and mechanisms of mitochondrial transfer in Alzheimer’s disease and how these transfer activities contribute to disease progression.https://www.frontiersin.org/articles/10.3389/fnagi.2024.1517965/fullAlzheimer’s diseasemitochondrial transfermitochondrial dysfunctionneuroprotectionAD treatment
spellingShingle Yun Wei
Xinlei Du
Hongling Guo
Jingjing Han
Meixia Liu
Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transfer
Frontiers in Aging Neuroscience
Alzheimer’s disease
mitochondrial transfer
mitochondrial dysfunction
neuroprotection
AD treatment
title Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transfer
title_full Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transfer
title_fullStr Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transfer
title_full_unstemmed Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transfer
title_short Mitochondrial dysfunction and Alzheimer’s disease: pathogenesis of mitochondrial transfer
title_sort mitochondrial dysfunction and alzheimer s disease pathogenesis of mitochondrial transfer
topic Alzheimer’s disease
mitochondrial transfer
mitochondrial dysfunction
neuroprotection
AD treatment
url https://www.frontiersin.org/articles/10.3389/fnagi.2024.1517965/full
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AT xinleidu mitochondrialdysfunctionandalzheimersdiseasepathogenesisofmitochondrialtransfer
AT honglingguo mitochondrialdysfunctionandalzheimersdiseasepathogenesisofmitochondrialtransfer
AT jingjinghan mitochondrialdysfunctionandalzheimersdiseasepathogenesisofmitochondrialtransfer
AT meixialiu mitochondrialdysfunctionandalzheimersdiseasepathogenesisofmitochondrialtransfer