Biomarkers of Inflammation in Obesity-Psoriatic Patients

Psoriasis is a common chronic inflammatory multisystemic disease with a complex pathogenesis consisting of genetic, immunological, and environmental components. It is associated with a number of comorbidities, including diabetes, metabolic syndrome, obesity, and myocardial infarction. In addition, t...

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Main Authors: Carmen Rodríguez-Cerdeira, Mónica Cordeiro-Rodríguez, Miguel Carnero-Gregorio, Adriana López-Barcenas, Erick Martínez-Herrera, Gabriella Fabbrocini, Ardiana Sinani, Roberto Arenas-Guzmán, José Luís González-Cespón
Format: Article
Language:English
Published: Wiley 2019-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2019/7353420
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author Carmen Rodríguez-Cerdeira
Mónica Cordeiro-Rodríguez
Miguel Carnero-Gregorio
Adriana López-Barcenas
Erick Martínez-Herrera
Gabriella Fabbrocini
Ardiana Sinani
Roberto Arenas-Guzmán
José Luís González-Cespón
author_facet Carmen Rodríguez-Cerdeira
Mónica Cordeiro-Rodríguez
Miguel Carnero-Gregorio
Adriana López-Barcenas
Erick Martínez-Herrera
Gabriella Fabbrocini
Ardiana Sinani
Roberto Arenas-Guzmán
José Luís González-Cespón
author_sort Carmen Rodríguez-Cerdeira
collection DOAJ
description Psoriasis is a common chronic inflammatory multisystemic disease with a complex pathogenesis consisting of genetic, immunological, and environmental components. It is associated with a number of comorbidities, including diabetes, metabolic syndrome, obesity, and myocardial infarction. In addition, the severity of psoriasis seems to be related to the severity of obesity. Patients with higher levels of obesity show poorer response to systemic treatments of psoriasis. Several studies have demonstrated that white adipose tissue is a crucial site of the formation of proinflammatory adipokines such as leptin, adiponectin, and resistin and classical cytokines such as interleukin- (IL-) 6 and tumour necrosis factor-α. In psoriasis, due to the proliferation of Th1, Th17, and Th22 cells, IL-22, among others, is produced in addition to the abovementioned cytokines. With respect to leptin and resistin, both of these adipokines are present in high levels in obese persons with psoriasis. Further, the plasma levels of leptin and resistin are related to the severity of psoriasis. These results strongly suggest that obesity, through proinflammatory pathways, is a predisposing factor to the development of psoriasis and that obesity aggravates existing psoriasis. Different inflammatory biomarkers link psoriasis and obesity. In this paper, the most important ones are described.
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publisher Wiley
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series Mediators of Inflammation
spelling doaj-art-19d92e2f9e6b42a7a73574daae0fdb902025-08-20T02:02:08ZengWileyMediators of Inflammation0962-93511466-18612019-01-01201910.1155/2019/73534207353420Biomarkers of Inflammation in Obesity-Psoriatic PatientsCarmen Rodríguez-Cerdeira0Mónica Cordeiro-Rodríguez1Miguel Carnero-Gregorio2Adriana López-Barcenas3Erick Martínez-Herrera4Gabriella Fabbrocini5Ardiana Sinani6Roberto Arenas-Guzmán7José Luís González-Cespón8Efficiency, Quality and Costs in Health Services Research Group (EFISALUD), Health Research Institute, SERGAS-UVIGO, Vigo, SpainEfficiency, Quality and Costs in Health Services Research Group (EFISALUD), Health Research Institute, SERGAS-UVIGO, Vigo, SpainEfficiency, Quality and Costs in Health Services Research Group (EFISALUD), Health Research Institute, SERGAS-UVIGO, Vigo, SpainEuropean Women Dermatological and Venereological Society (EWDVS), Vigo, SpainPsychodermatology Task Force of the Ibero Latin American College of Dermatology, ArgentinaEuropean Women Dermatological and Venereological Society (EWDVS), Vigo, SpainEuropean Women Dermatological and Venereological Society (EWDVS), Vigo, SpainEuropean Women Dermatological and Venereological Society (EWDVS), Vigo, SpainEfficiency, Quality and Costs in Health Services Research Group (EFISALUD), Health Research Institute, SERGAS-UVIGO, Vigo, SpainPsoriasis is a common chronic inflammatory multisystemic disease with a complex pathogenesis consisting of genetic, immunological, and environmental components. It is associated with a number of comorbidities, including diabetes, metabolic syndrome, obesity, and myocardial infarction. In addition, the severity of psoriasis seems to be related to the severity of obesity. Patients with higher levels of obesity show poorer response to systemic treatments of psoriasis. Several studies have demonstrated that white adipose tissue is a crucial site of the formation of proinflammatory adipokines such as leptin, adiponectin, and resistin and classical cytokines such as interleukin- (IL-) 6 and tumour necrosis factor-α. In psoriasis, due to the proliferation of Th1, Th17, and Th22 cells, IL-22, among others, is produced in addition to the abovementioned cytokines. With respect to leptin and resistin, both of these adipokines are present in high levels in obese persons with psoriasis. Further, the plasma levels of leptin and resistin are related to the severity of psoriasis. These results strongly suggest that obesity, through proinflammatory pathways, is a predisposing factor to the development of psoriasis and that obesity aggravates existing psoriasis. Different inflammatory biomarkers link psoriasis and obesity. In this paper, the most important ones are described.http://dx.doi.org/10.1155/2019/7353420
spellingShingle Carmen Rodríguez-Cerdeira
Mónica Cordeiro-Rodríguez
Miguel Carnero-Gregorio
Adriana López-Barcenas
Erick Martínez-Herrera
Gabriella Fabbrocini
Ardiana Sinani
Roberto Arenas-Guzmán
José Luís González-Cespón
Biomarkers of Inflammation in Obesity-Psoriatic Patients
Mediators of Inflammation
title Biomarkers of Inflammation in Obesity-Psoriatic Patients
title_full Biomarkers of Inflammation in Obesity-Psoriatic Patients
title_fullStr Biomarkers of Inflammation in Obesity-Psoriatic Patients
title_full_unstemmed Biomarkers of Inflammation in Obesity-Psoriatic Patients
title_short Biomarkers of Inflammation in Obesity-Psoriatic Patients
title_sort biomarkers of inflammation in obesity psoriatic patients
url http://dx.doi.org/10.1155/2019/7353420
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