Irisin Attenuates Neuroinflammation Targeting the NLRP3 Inflammasome

Neuroinflammation is defined as an immune response involving various cell types, particularly microglia, which monitor the neuroimmune axis. Microglia activate in two distinct ways: M1, which is pro-inflammatory and capable of inducing phagocytosis and releasing pro-inflammatory factors, and M2, whi...

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Main Authors: Francesca Martina Filannino, Melania Ruggiero, Maria Antonietta Panaro, Dario Domenico Lofrumento, Teresa Trotta, Tarek Benameur, Antonia Cianciulli, Rosa Calvello, Federico Zoila, Chiara Porro
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Language:English
Published: MDPI AG 2024-11-01
Series:Molecules
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Online Access:https://www.mdpi.com/1420-3049/29/23/5623
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author Francesca Martina Filannino
Melania Ruggiero
Maria Antonietta Panaro
Dario Domenico Lofrumento
Teresa Trotta
Tarek Benameur
Antonia Cianciulli
Rosa Calvello
Federico Zoila
Chiara Porro
author_facet Francesca Martina Filannino
Melania Ruggiero
Maria Antonietta Panaro
Dario Domenico Lofrumento
Teresa Trotta
Tarek Benameur
Antonia Cianciulli
Rosa Calvello
Federico Zoila
Chiara Porro
author_sort Francesca Martina Filannino
collection DOAJ
description Neuroinflammation is defined as an immune response involving various cell types, particularly microglia, which monitor the neuroimmune axis. Microglia activate in two distinct ways: M1, which is pro-inflammatory and capable of inducing phagocytosis and releasing pro-inflammatory factors, and M2, which has anti-inflammatory properties. Inflammasomes are large protein complexes that form in response to internal danger signals, activating caspase-1 and leading to the release of pro-inflammatory cytokines such as interleukin 1β. Irisin, a peptide primarily released by muscles during exercise, was examined for its effects on BV2 microglial cells in vitro. Even at low concentrations, irisin was observed to influence the NLRP3 inflammasome, showing potential as a neuroprotective and anti-inflammatory agent after stimulation with lipopolysaccharides (LPSs). Irisin helped maintain microglia in their typical physiological state and reduced their migratory capacity. Irisin also increased Arg-1 protein expression, a marker of M2 polarization, while downregulating NLRP3, Pycard, caspase-1, IL-1β, and CD14. The results of this study indicate that irisin may serve as a crucial mediator of neuroprotection, thus representing an innovative tool for the prevention of neurodegenerative diseases.
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spelling doaj-art-164fdc768c764bf0ac8ccbb23c697e132025-08-20T02:50:38ZengMDPI AGMolecules1420-30492024-11-012923562310.3390/molecules29235623Irisin Attenuates Neuroinflammation Targeting the NLRP3 InflammasomeFrancesca Martina Filannino0Melania Ruggiero1Maria Antonietta Panaro2Dario Domenico Lofrumento3Teresa Trotta4Tarek Benameur5Antonia Cianciulli6Rosa Calvello7Federico Zoila8Chiara Porro9Department of Clinical and Experimental Medicine, University of Foggia, I-71100 Foggia, ItalyDepartment of Biosciences, Biotechnologies and Environment, University of Bari, I-70125 Bari, ItalyDepartment of Biosciences, Biotechnologies and Environment, University of Bari, I-70125 Bari, ItalyDepartment of Biological and Environmental Sciences and Technologies, Section of Human Anatomy, University of Salento, I-73100 Lecce, ItalyDepartment of Clinical and Experimental Medicine, University of Foggia, I-71100 Foggia, ItalyDepartment of Biomedical Sciences, College of Medicine, King Faisal University, Al-Ahsa 31982, Saudi ArabiaDepartment of Biosciences, Biotechnologies and Environment, University of Bari, I-70125 Bari, ItalyDepartment of Biosciences, Biotechnologies and Environment, University of Bari, I-70125 Bari, ItalyDepartment of Clinical and Experimental Medicine, University of Foggia, I-71100 Foggia, ItalyDepartment of Clinical and Experimental Medicine, University of Foggia, I-71100 Foggia, ItalyNeuroinflammation is defined as an immune response involving various cell types, particularly microglia, which monitor the neuroimmune axis. Microglia activate in two distinct ways: M1, which is pro-inflammatory and capable of inducing phagocytosis and releasing pro-inflammatory factors, and M2, which has anti-inflammatory properties. Inflammasomes are large protein complexes that form in response to internal danger signals, activating caspase-1 and leading to the release of pro-inflammatory cytokines such as interleukin 1β. Irisin, a peptide primarily released by muscles during exercise, was examined for its effects on BV2 microglial cells in vitro. Even at low concentrations, irisin was observed to influence the NLRP3 inflammasome, showing potential as a neuroprotective and anti-inflammatory agent after stimulation with lipopolysaccharides (LPSs). Irisin helped maintain microglia in their typical physiological state and reduced their migratory capacity. Irisin also increased Arg-1 protein expression, a marker of M2 polarization, while downregulating NLRP3, Pycard, caspase-1, IL-1β, and CD14. The results of this study indicate that irisin may serve as a crucial mediator of neuroprotection, thus representing an innovative tool for the prevention of neurodegenerative diseases.https://www.mdpi.com/1420-3049/29/23/5623microgliairisininflammasomeneuroinflammation
spellingShingle Francesca Martina Filannino
Melania Ruggiero
Maria Antonietta Panaro
Dario Domenico Lofrumento
Teresa Trotta
Tarek Benameur
Antonia Cianciulli
Rosa Calvello
Federico Zoila
Chiara Porro
Irisin Attenuates Neuroinflammation Targeting the NLRP3 Inflammasome
Molecules
microglia
irisin
inflammasome
neuroinflammation
title Irisin Attenuates Neuroinflammation Targeting the NLRP3 Inflammasome
title_full Irisin Attenuates Neuroinflammation Targeting the NLRP3 Inflammasome
title_fullStr Irisin Attenuates Neuroinflammation Targeting the NLRP3 Inflammasome
title_full_unstemmed Irisin Attenuates Neuroinflammation Targeting the NLRP3 Inflammasome
title_short Irisin Attenuates Neuroinflammation Targeting the NLRP3 Inflammasome
title_sort irisin attenuates neuroinflammation targeting the nlrp3 inflammasome
topic microglia
irisin
inflammasome
neuroinflammation
url https://www.mdpi.com/1420-3049/29/23/5623
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