Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)

Background: pl6 (CDKN2a) play a role in tumorigenesis in some head and neck squamous cell carcinomas. Frequent homozygous deletions of the pl6 gene have been reported in many tumor cell lines including the brain, breast, osteosarcomas, melanomas, kidney, bladder, and ovary. Materials and methods: 5...

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Main Authors: Neetu Oommen, Vasanthi V, Ramya Ramadoss, Rajkumar Krishnan
Format: Article
Language:English
Published: Elsevier 2024-09-01
Series:Oral Oncology Reports
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Online Access:http://www.sciencedirect.com/science/article/pii/S2772906024004783
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author Neetu Oommen
Vasanthi V
Ramya Ramadoss
Rajkumar Krishnan
author_facet Neetu Oommen
Vasanthi V
Ramya Ramadoss
Rajkumar Krishnan
author_sort Neetu Oommen
collection DOAJ
description Background: pl6 (CDKN2a) play a role in tumorigenesis in some head and neck squamous cell carcinomas. Frequent homozygous deletions of the pl6 gene have been reported in many tumor cell lines including the brain, breast, osteosarcomas, melanomas, kidney, bladder, and ovary. Materials and methods: 5 patients without any tobacco using habit were included in group I as controls. 10 clinically and histopathologically confirmed cases of oral potentially malignant disorders (OPMDs) [oral submucous fibrosis (OSMF) −7 & leukoplakia (moderate dysplasia) −3] were included in group II. 10 clinically and histopathologically confirmed cases of OSCC were categorized as group III. Buccal scrapings were taken and analyzed for exon 1, 2, 3 of p16 and homozygous deletion in exon 2 of p16 was also detected by PCR and gel electrophoresis. Results: PCR amplification products of exon 1, 2, 3 of p16 were found in all 25 samples which include 10 cases of OSCC, 10 cases of OPMDs and 5 controls. Homozygous deletion in exon 2 was found only in 30 % of OSCC and 20 % of OPMD. Conclusion: Our study showed that cytological samples yield sufficient amount of DNA, which showed 100 % positivity with exon 1, 2, 3 of p16 gene, but the percentage of genetic alteration of p16 gene seems to be less when compared with other related studies.
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spelling doaj-art-143bfa184b1e41ef933a62d8864626dc2025-08-20T02:44:32ZengElsevierOral Oncology Reports2772-90602024-09-011110063210.1016/j.oor.2024.100632Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)Neetu Oommen0Vasanthi V1Ramya Ramadoss2Rajkumar Krishnan3Department of Oral Pathology and Microbiology, SRM Dental College, Bharathi salai, Chennai, IndiaDepartment of Oral Pathology and Microbiology, SRM Dental College, Bharathi salai, Chennai, India; Corresponding author. Department of Oral Pathology & Microbiology, SRM Dental College, Bharathi salai, Chennai, India.Department of Oral Biology, Saveetha Dental College, Velappanchavadi, Chennai, IndiaDepartment of Oral Pathology and Microbiology, SRM Dental College, Bharathi salai, Chennai, IndiaBackground: pl6 (CDKN2a) play a role in tumorigenesis in some head and neck squamous cell carcinomas. Frequent homozygous deletions of the pl6 gene have been reported in many tumor cell lines including the brain, breast, osteosarcomas, melanomas, kidney, bladder, and ovary. Materials and methods: 5 patients without any tobacco using habit were included in group I as controls. 10 clinically and histopathologically confirmed cases of oral potentially malignant disorders (OPMDs) [oral submucous fibrosis (OSMF) −7 & leukoplakia (moderate dysplasia) −3] were included in group II. 10 clinically and histopathologically confirmed cases of OSCC were categorized as group III. Buccal scrapings were taken and analyzed for exon 1, 2, 3 of p16 and homozygous deletion in exon 2 of p16 was also detected by PCR and gel electrophoresis. Results: PCR amplification products of exon 1, 2, 3 of p16 were found in all 25 samples which include 10 cases of OSCC, 10 cases of OPMDs and 5 controls. Homozygous deletion in exon 2 was found only in 30 % of OSCC and 20 % of OPMD. Conclusion: Our study showed that cytological samples yield sufficient amount of DNA, which showed 100 % positivity with exon 1, 2, 3 of p16 gene, but the percentage of genetic alteration of p16 gene seems to be less when compared with other related studies.http://www.sciencedirect.com/science/article/pii/S2772906024004783Cytologyp16OPMDOSCCsingle nucleotide polymorphism
spellingShingle Neetu Oommen
Vasanthi V
Ramya Ramadoss
Rajkumar Krishnan
Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)
Oral Oncology Reports
Cytology
p16
OPMD
OSCC
single nucleotide polymorphism
title Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)
title_full Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)
title_fullStr Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)
title_full_unstemmed Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)
title_short Analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders (OPMD) and oral squamous cell carcinoma (OSCC)
title_sort analysis of single nucleotide polymorphism of p16 gene in cytological samples of patients with oral potentially malignant disorders opmd and oral squamous cell carcinoma oscc
topic Cytology
p16
OPMD
OSCC
single nucleotide polymorphism
url http://www.sciencedirect.com/science/article/pii/S2772906024004783
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