Screening assays for heart function mutants in Drosophila

The rapid life cycle and genetic tractability of Drosophila make it an ideal organism for large-scale genetic screens. Here we describe a novel assay for pupal heart rate and rhythmicity, as well as techniques to measure adult cardiac stress response. These assays can be powerfully combined to concu...

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Main Authors: Robert J. Wessells, Rolf Bodmer
Format: Article
Language:English
Published: Taylor & Francis Group 2004-07-01
Series:BioTechniques
Online Access:https://www.future-science.com/doi/10.2144/04371ST01
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author Robert J. Wessells
Rolf Bodmer
author_facet Robert J. Wessells
Rolf Bodmer
author_sort Robert J. Wessells
collection DOAJ
description The rapid life cycle and genetic tractability of Drosophila make it an ideal organism for large-scale genetic screens. Here we describe a novel assay for pupal heart rate and rhythmicity, as well as techniques to measure adult cardiac stress response. These assays can be powerfully combined to concurrently screen for both mutations affecting cardiac function and mutations affecting the age-dependent decline in adult cardiac stress response. Mutations identified in such screens have the potential to contribute greatly to the understanding of both congenital heart disease and the regulation of age-dependent decline in cardiac function in the human population.
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1940-9818
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spelling doaj-art-14300df035724dbc93bb82cf18f4eaf22025-08-20T02:26:09ZengTaylor & Francis GroupBioTechniques0736-62051940-98182004-07-01371586610.2144/04371ST01Screening assays for heart function mutants in DrosophilaRobert J. Wessells0Rolf Bodmer11The Burnham Institute, La Jolla, CA, USA1The Burnham Institute, La Jolla, CA, USAThe rapid life cycle and genetic tractability of Drosophila make it an ideal organism for large-scale genetic screens. Here we describe a novel assay for pupal heart rate and rhythmicity, as well as techniques to measure adult cardiac stress response. These assays can be powerfully combined to concurrently screen for both mutations affecting cardiac function and mutations affecting the age-dependent decline in adult cardiac stress response. Mutations identified in such screens have the potential to contribute greatly to the understanding of both congenital heart disease and the regulation of age-dependent decline in cardiac function in the human population.https://www.future-science.com/doi/10.2144/04371ST01
spellingShingle Robert J. Wessells
Rolf Bodmer
Screening assays for heart function mutants in Drosophila
BioTechniques
title Screening assays for heart function mutants in Drosophila
title_full Screening assays for heart function mutants in Drosophila
title_fullStr Screening assays for heart function mutants in Drosophila
title_full_unstemmed Screening assays for heart function mutants in Drosophila
title_short Screening assays for heart function mutants in Drosophila
title_sort screening assays for heart function mutants in drosophila
url https://www.future-science.com/doi/10.2144/04371ST01
work_keys_str_mv AT robertjwessells screeningassaysforheartfunctionmutantsindrosophila
AT rolfbodmer screeningassaysforheartfunctionmutantsindrosophila