Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardation

Background. Alterations in blood coagulation and fibrinolysis are believed to play an important role in the pathogenesis of preeclampsia. Hypercoagulability may be associated with features seen in preeclampsia, such as fibrin deposition in various organs, consumptive thrombocytopenia, and placental...

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Main Authors: Rune Schjetlein, Guttorm Haugen, Finn Wisløff
Format: Article
Language:English
Published: Wiley 1997-06-01
Series:Acta Obstetricia et Gynecologica Scandinavica
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Online Access:https://doi.org/10.3109/00016349709024580
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author Rune Schjetlein
Guttorm Haugen
Finn Wisløff
author_facet Rune Schjetlein
Guttorm Haugen
Finn Wisløff
author_sort Rune Schjetlein
collection DOAJ
description Background. Alterations in blood coagulation and fibrinolysis are believed to play an important role in the pathogenesis of preeclampsia. Hypercoagulability may be associated with features seen in preeclampsia, such as fibrin deposition in various organs, consumptive thrombocytopenia, and placental hypoperfusion, insufficiency and infarction. Methods. In this cohort study, we compared the plasma levels of markers of blood coagulation and fibrinolysis in preeclamptic women to normotensive, pregnant controls. We also studied the association between these markers and intrauterine growth retardation (IUGR). Results. In both mild and severe preeclampsia, the mean plasma concentrations of thrombin‐antithrombin III complex (TAT) and plasminogen activator inhibitor type 1 (PAI‐1) activity were significantly increased, while fibrinogen, antithrombin III (ATIII) and plasminogen activator inhibitor type‐2 (PAI‐2) antigen levels were significantly reduced compared to controls. Plasma D‐dimer concentration was significantly higher in severe, but not in mild preeclampsia compared to the controls. The plasma level of fibrin monomer was similar in patients and controls. Patients with preeclampsia delivering growth retarded infants had significantly lower PAI‐1 activity and PAI‐2 antigen concentrations in plasma than the remaining preeclamptic women. Conclusions. We found evidence of increased intravascular coagulation and fibrin turnover in preeclampsia. Low PAI‐2 antigen plasma levels were associated with severe preeclampsia and IUGR.
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spelling doaj-art-13c5d1104ceb436bbe4f2b5df104f0a02025-08-20T02:37:32ZengWileyActa Obstetricia et Gynecologica Scandinavica0001-63491600-04121997-06-0176654154610.3109/00016349709024580Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardationRune Schjetlein0Guttorm Haugen1Finn Wisløff2Hematological Research Laboratory, Ullevål Hospital, NorwayDepartment of Obstetrics and Gynecology, The National Hospital, University of Oslo, NorwayHematological Research Laboratory, Ullevål Hospital, NorwayBackground. Alterations in blood coagulation and fibrinolysis are believed to play an important role in the pathogenesis of preeclampsia. Hypercoagulability may be associated with features seen in preeclampsia, such as fibrin deposition in various organs, consumptive thrombocytopenia, and placental hypoperfusion, insufficiency and infarction. Methods. In this cohort study, we compared the plasma levels of markers of blood coagulation and fibrinolysis in preeclamptic women to normotensive, pregnant controls. We also studied the association between these markers and intrauterine growth retardation (IUGR). Results. In both mild and severe preeclampsia, the mean plasma concentrations of thrombin‐antithrombin III complex (TAT) and plasminogen activator inhibitor type 1 (PAI‐1) activity were significantly increased, while fibrinogen, antithrombin III (ATIII) and plasminogen activator inhibitor type‐2 (PAI‐2) antigen levels were significantly reduced compared to controls. Plasma D‐dimer concentration was significantly higher in severe, but not in mild preeclampsia compared to the controls. The plasma level of fibrin monomer was similar in patients and controls. Patients with preeclampsia delivering growth retarded infants had significantly lower PAI‐1 activity and PAI‐2 antigen concentrations in plasma than the remaining preeclamptic women. Conclusions. We found evidence of increased intravascular coagulation and fibrin turnover in preeclampsia. Low PAI‐2 antigen plasma levels were associated with severe preeclampsia and IUGR.https://doi.org/10.3109/00016349709024580coagulationfibrinolysisintrauterine growth retardationpreeclampsia
spellingShingle Rune Schjetlein
Guttorm Haugen
Finn Wisløff
Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardation
Acta Obstetricia et Gynecologica Scandinavica
coagulation
fibrinolysis
intrauterine growth retardation
preeclampsia
title Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardation
title_full Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardation
title_fullStr Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardation
title_full_unstemmed Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardation
title_short Markers of intravascular coagulation and fibrinolysis in preeclampsia: association with intrauterine growth retardation
title_sort markers of intravascular coagulation and fibrinolysis in preeclampsia association with intrauterine growth retardation
topic coagulation
fibrinolysis
intrauterine growth retardation
preeclampsia
url https://doi.org/10.3109/00016349709024580
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AT guttormhaugen markersofintravascularcoagulationandfibrinolysisinpreeclampsiaassociationwithintrauterinegrowthretardation
AT finnwisløff markersofintravascularcoagulationandfibrinolysisinpreeclampsiaassociationwithintrauterinegrowthretardation