C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function

Abstract C3G, a Rap1 GEF, promotes megakaryopoiesis and platelet function. Using transgenic and knock-out mouse models targeting C3G in megakaryocytes, we investigated whether C3G also affects the niche function of megakaryocytes during bone marrow (BM) recovery after myeloablation induced by 5-fluo...

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Main Authors: Óscar Herranz, Pablo Berrocal, Carmen Sicilia-Navarro, Cristina Fernández-Infante, Luis Hernández-Cano, Almudena Porras, Carmen Guerrero
Format: Article
Language:English
Published: BMC 2025-04-01
Series:Journal of Hematology & Oncology
Subjects:
Online Access:https://doi.org/10.1186/s13045-025-01687-1
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author Óscar Herranz
Pablo Berrocal
Carmen Sicilia-Navarro
Cristina Fernández-Infante
Luis Hernández-Cano
Almudena Porras
Carmen Guerrero
author_facet Óscar Herranz
Pablo Berrocal
Carmen Sicilia-Navarro
Cristina Fernández-Infante
Luis Hernández-Cano
Almudena Porras
Carmen Guerrero
author_sort Óscar Herranz
collection DOAJ
description Abstract C3G, a Rap1 GEF, promotes megakaryopoiesis and platelet function. Using transgenic and knock-out mouse models targeting C3G in megakaryocytes, we investigated whether C3G also affects the niche function of megakaryocytes during bone marrow (BM) recovery after myeloablation induced by 5-fluorouracil (5-FU), or total body irradiation (TBI) followed by bone marrow transplantation. C3G promoted megakaryocyte maturation and platelet production during recovery, along with increased white and red blood cell counts and enhanced survival of female mice after repeated doses of 5-FU. Additionally, megakaryocytes favored adipocyte differentiation through a C3G-mediated mechanism, likely involving Fgf1. Changes in the number or behavior of BM megakaryocytes and adipocytes influenced the hematopoietic stem cell pool, with C3G promoting its bias towards the myeloid-megakaryocytic lineage in both 5-FU- and TBI-ablated models. Therefore, C3G could be a potential target in therapies aimed at enhancing hematopoiesis in patients undergoing chemotherapy and/or BM transplantation.
format Article
id doaj-art-129f2b034b6f420db8586ec82ef9c7a7
institution OA Journals
issn 1756-8722
language English
publishDate 2025-04-01
publisher BMC
record_format Article
series Journal of Hematology & Oncology
spelling doaj-art-129f2b034b6f420db8586ec82ef9c7a72025-08-20T01:54:19ZengBMCJournal of Hematology & Oncology1756-87222025-04-0118112010.1186/s13045-025-01687-1C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche functionÓscar Herranz0Pablo Berrocal1Carmen Sicilia-Navarro2Cristina Fernández-Infante3Luis Hernández-Cano4Almudena Porras5Carmen Guerrero6Centro de Investigación del Cáncer (CIC), USAL-CSICCentro de Investigación del Cáncer (CIC), USAL-CSICCentro de Investigación del Cáncer (CIC), USAL-CSICCentro de Investigación del Cáncer (CIC), USAL-CSICCentro de Investigación del Cáncer (CIC), USAL-CSICDepartamento de Bioquímica y Biología Molecular, Facultad de Farmacia, Universidad Complutense de MadridCentro de Investigación del Cáncer (CIC), USAL-CSICAbstract C3G, a Rap1 GEF, promotes megakaryopoiesis and platelet function. Using transgenic and knock-out mouse models targeting C3G in megakaryocytes, we investigated whether C3G also affects the niche function of megakaryocytes during bone marrow (BM) recovery after myeloablation induced by 5-fluorouracil (5-FU), or total body irradiation (TBI) followed by bone marrow transplantation. C3G promoted megakaryocyte maturation and platelet production during recovery, along with increased white and red blood cell counts and enhanced survival of female mice after repeated doses of 5-FU. Additionally, megakaryocytes favored adipocyte differentiation through a C3G-mediated mechanism, likely involving Fgf1. Changes in the number or behavior of BM megakaryocytes and adipocytes influenced the hematopoietic stem cell pool, with C3G promoting its bias towards the myeloid-megakaryocytic lineage in both 5-FU- and TBI-ablated models. Therefore, C3G could be a potential target in therapies aimed at enhancing hematopoiesis in patients undergoing chemotherapy and/or BM transplantation.https://doi.org/10.1186/s13045-025-01687-1Rapgef1MegakaryocyteBone marrow nicheMyeloablationAdipocyteHematopoietic stem cell
spellingShingle Óscar Herranz
Pablo Berrocal
Carmen Sicilia-Navarro
Cristina Fernández-Infante
Luis Hernández-Cano
Almudena Porras
Carmen Guerrero
C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function
Journal of Hematology & Oncology
Rapgef1
Megakaryocyte
Bone marrow niche
Myeloablation
Adipocyte
Hematopoietic stem cell
title C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function
title_full C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function
title_fullStr C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function
title_full_unstemmed C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function
title_short C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function
title_sort c3g promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function
topic Rapgef1
Megakaryocyte
Bone marrow niche
Myeloablation
Adipocyte
Hematopoietic stem cell
url https://doi.org/10.1186/s13045-025-01687-1
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