Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injury

Abstract Sepsis is a syndrome of organ dysfunction caused by the invasion of pathogenic microorganisms. In clinical practice, patients with sepsis are prone to concurrent acute kidney injury, which has high morbidity and mortality rates. Thus, understanding the pathogenesis of sepsis-associated acut...

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Main Authors: Yuhang Yang, Xinqi Deng, Wenyuan Li, Yan Leng, Yonghong Xiong, Bihan Wang, Siyuan Gong, Yunhao Wang, Baichuan Yang, Wei Li
Format: Article
Language:English
Published: BMC 2025-04-01
Series:Clinical Epigenetics
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Online Access:https://doi.org/10.1186/s13148-025-01861-9
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author Yuhang Yang
Xinqi Deng
Wenyuan Li
Yan Leng
Yonghong Xiong
Bihan Wang
Siyuan Gong
Yunhao Wang
Baichuan Yang
Wei Li
author_facet Yuhang Yang
Xinqi Deng
Wenyuan Li
Yan Leng
Yonghong Xiong
Bihan Wang
Siyuan Gong
Yunhao Wang
Baichuan Yang
Wei Li
author_sort Yuhang Yang
collection DOAJ
description Abstract Sepsis is a syndrome of organ dysfunction caused by the invasion of pathogenic microorganisms. In clinical practice, patients with sepsis are prone to concurrent acute kidney injury, which has high morbidity and mortality rates. Thus, understanding the pathogenesis of sepsis-associated acute kidney injury is of significant clinical importance. Ferroptosis is an iron-dependent programmed cell death pathway, which is proved to play a critical role in the process of sepsis-associated acute kidney injury through various mechanisms. Epigenetic regulation modulates the content and function of nucleic acids and proteins within cells through various modifications. Its impact on ferroptosis has garnered increasing attention; however, the role of epigenetic regulation targeting ferroptosis in sepsis-associated acute kidney injury has not been fully elucidated. Growing evidence suggests that epigenetic regulation can modulate ferroptosis through complex pathway networks, thereby affecting the development and prognosis of sepsis-associated acute kidney injury. This paper summarizes the impact of ferroptosis on sepsis-associated acute kidney injury and the regulatory mechanisms of epigenetic regulation on ferroptosis, providing new insights for the targeted therapy of sepsis-associated acute kidney injury.
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issn 1868-7083
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publisher BMC
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series Clinical Epigenetics
spelling doaj-art-1278c8fefad34bf28d96c65cbde8f98c2025-08-20T03:10:06ZengBMCClinical Epigenetics1868-70832025-04-0117111410.1186/s13148-025-01861-9Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injuryYuhang Yang0Xinqi Deng1Wenyuan Li2Yan Leng3Yonghong Xiong4Bihan Wang5Siyuan Gong6Yunhao Wang7Baichuan Yang8Wei Li9Department of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityDepartment of Anesthesiology, Renmin Hospital of Wuhan UniversityAbstract Sepsis is a syndrome of organ dysfunction caused by the invasion of pathogenic microorganisms. In clinical practice, patients with sepsis are prone to concurrent acute kidney injury, which has high morbidity and mortality rates. Thus, understanding the pathogenesis of sepsis-associated acute kidney injury is of significant clinical importance. Ferroptosis is an iron-dependent programmed cell death pathway, which is proved to play a critical role in the process of sepsis-associated acute kidney injury through various mechanisms. Epigenetic regulation modulates the content and function of nucleic acids and proteins within cells through various modifications. Its impact on ferroptosis has garnered increasing attention; however, the role of epigenetic regulation targeting ferroptosis in sepsis-associated acute kidney injury has not been fully elucidated. Growing evidence suggests that epigenetic regulation can modulate ferroptosis through complex pathway networks, thereby affecting the development and prognosis of sepsis-associated acute kidney injury. This paper summarizes the impact of ferroptosis on sepsis-associated acute kidney injury and the regulatory mechanisms of epigenetic regulation on ferroptosis, providing new insights for the targeted therapy of sepsis-associated acute kidney injury.https://doi.org/10.1186/s13148-025-01861-9Acute kidney injurySepsisFerroptosisEpigenetic regulation
spellingShingle Yuhang Yang
Xinqi Deng
Wenyuan Li
Yan Leng
Yonghong Xiong
Bihan Wang
Siyuan Gong
Yunhao Wang
Baichuan Yang
Wei Li
Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injury
Clinical Epigenetics
Acute kidney injury
Sepsis
Ferroptosis
Epigenetic regulation
title Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injury
title_full Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injury
title_fullStr Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injury
title_full_unstemmed Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injury
title_short Targeting the epigenetic regulation of ferroptosis: a potential therapeutic approach for sepsis-associated acute kidney injury
title_sort targeting the epigenetic regulation of ferroptosis a potential therapeutic approach for sepsis associated acute kidney injury
topic Acute kidney injury
Sepsis
Ferroptosis
Epigenetic regulation
url https://doi.org/10.1186/s13148-025-01861-9
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