The Association between E326K of GBA and the Risk of Parkinson’s Disease

It is reported that both the homozygous and heterozygous states of GBA mutations which are the causes of Gaucher disease (GD) are linked to the risk of PD. However, the GBA variant p.E326K (c.1093G > A, rs2230288), which does not result in GD in homozygous carriers, has triggered debate among exp...

Full description

Saved in:
Bibliographic Details
Main Authors: Yongpan Huang, Langmei Deng, Yanjun Zhong, Minhan Yi
Format: Article
Language:English
Published: Wiley 2018-01-01
Series:Parkinson's Disease
Online Access:http://dx.doi.org/10.1155/2018/1048084
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850236749538656256
author Yongpan Huang
Langmei Deng
Yanjun Zhong
Minhan Yi
author_facet Yongpan Huang
Langmei Deng
Yanjun Zhong
Minhan Yi
author_sort Yongpan Huang
collection DOAJ
description It is reported that both the homozygous and heterozygous states of GBA mutations which are the causes of Gaucher disease (GD) are linked to the risk of PD. However, the GBA variant p.E326K (c.1093G > A, rs2230288), which does not result in GD in homozygous carriers, has triggered debate among experts studying Parkinson's disease (PD). In order to determine if the E326K variant of GBA is associated with the risk of PD, a standard meta-analysis was conducted by searching and screening publications, data extraction, and statistical analysis. Finally, a total of 15 publications, containing 5,908 PD patients and 5,605 controls, were included in this analysis. The pooled OR of the E326K genotype analysis was 1.99 (95% CI: 1.57–2.51). The minor allele frequencies of E326K for PD patients and controls were 1.67% and 1.03%, respectively. The pooled OR for the minor allele A was 1.99 (95% CI: 1.58–2.50). According to the subgroup analysis, we found that the significant differences between PD patients and controls for both genotype and allele of E326K also exist in Asians and Caucasians, respectively. In this study, we found that E326K of GBA is associated with the risk of PD in total populations, Asians, and Caucasians, respectively. Further studies are needed to clarify the role of GBA in the pathogenesis of PD.
format Article
id doaj-art-1198331cf6834bdb83010e40a9453b59
institution OA Journals
issn 2090-8083
2042-0080
language English
publishDate 2018-01-01
publisher Wiley
record_format Article
series Parkinson's Disease
spelling doaj-art-1198331cf6834bdb83010e40a9453b592025-08-20T02:01:54ZengWileyParkinson's Disease2090-80832042-00802018-01-01201810.1155/2018/10480841048084The Association between E326K of GBA and the Risk of Parkinson’s DiseaseYongpan Huang0Langmei Deng1Yanjun Zhong2Minhan Yi3Information Security and Big Data Research Institute, Central South University, Changsha, Hunan, ChinaDepartment of Emergency, The Third Xiangya Hospital and School of Life Sciences, Central South University, Changsha, Hunan, ChinaICU Centre, The Second Xiangya Hospital, Central South University, Changsha, Hunan, ChinaInformation Security and Big Data Research Institute, Central South University, Changsha, Hunan, ChinaIt is reported that both the homozygous and heterozygous states of GBA mutations which are the causes of Gaucher disease (GD) are linked to the risk of PD. However, the GBA variant p.E326K (c.1093G > A, rs2230288), which does not result in GD in homozygous carriers, has triggered debate among experts studying Parkinson's disease (PD). In order to determine if the E326K variant of GBA is associated with the risk of PD, a standard meta-analysis was conducted by searching and screening publications, data extraction, and statistical analysis. Finally, a total of 15 publications, containing 5,908 PD patients and 5,605 controls, were included in this analysis. The pooled OR of the E326K genotype analysis was 1.99 (95% CI: 1.57–2.51). The minor allele frequencies of E326K for PD patients and controls were 1.67% and 1.03%, respectively. The pooled OR for the minor allele A was 1.99 (95% CI: 1.58–2.50). According to the subgroup analysis, we found that the significant differences between PD patients and controls for both genotype and allele of E326K also exist in Asians and Caucasians, respectively. In this study, we found that E326K of GBA is associated with the risk of PD in total populations, Asians, and Caucasians, respectively. Further studies are needed to clarify the role of GBA in the pathogenesis of PD.http://dx.doi.org/10.1155/2018/1048084
spellingShingle Yongpan Huang
Langmei Deng
Yanjun Zhong
Minhan Yi
The Association between E326K of GBA and the Risk of Parkinson’s Disease
Parkinson's Disease
title The Association between E326K of GBA and the Risk of Parkinson’s Disease
title_full The Association between E326K of GBA and the Risk of Parkinson’s Disease
title_fullStr The Association between E326K of GBA and the Risk of Parkinson’s Disease
title_full_unstemmed The Association between E326K of GBA and the Risk of Parkinson’s Disease
title_short The Association between E326K of GBA and the Risk of Parkinson’s Disease
title_sort association between e326k of gba and the risk of parkinson s disease
url http://dx.doi.org/10.1155/2018/1048084
work_keys_str_mv AT yongpanhuang theassociationbetweene326kofgbaandtheriskofparkinsonsdisease
AT langmeideng theassociationbetweene326kofgbaandtheriskofparkinsonsdisease
AT yanjunzhong theassociationbetweene326kofgbaandtheriskofparkinsonsdisease
AT minhanyi theassociationbetweene326kofgbaandtheriskofparkinsonsdisease
AT yongpanhuang associationbetweene326kofgbaandtheriskofparkinsonsdisease
AT langmeideng associationbetweene326kofgbaandtheriskofparkinsonsdisease
AT yanjunzhong associationbetweene326kofgbaandtheriskofparkinsonsdisease
AT minhanyi associationbetweene326kofgbaandtheriskofparkinsonsdisease