Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndrome

Abstract Background Ciliopathies are characterized by the dysfunction of cilia, being inherited retinal dystrophies (IRDs) included in sensory ciliopathies. Besides, oro-facial-digital syndrome (OFD) is caused by mutations in ciliary genes, leading to dysmorphic features. Mutations in TBC1D32 were a...

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Main Authors: Belén García-Bohórquez, Purificación Marín-Reina, Elena Aller, Pilar Barberán-Martínez, Miguel Armengot, Roberto Llorens-Salvador, Inmaculada Concepción Almor-Palacios, José M. Millán, Gema García-García
Format: Article
Language:English
Published: BMC 2025-05-01
Series:Human Genomics
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Online Access:https://doi.org/10.1186/s40246-025-00759-0
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author Belén García-Bohórquez
Purificación Marín-Reina
Elena Aller
Pilar Barberán-Martínez
Miguel Armengot
Roberto Llorens-Salvador
Inmaculada Concepción Almor-Palacios
José M. Millán
Gema García-García
author_facet Belén García-Bohórquez
Purificación Marín-Reina
Elena Aller
Pilar Barberán-Martínez
Miguel Armengot
Roberto Llorens-Salvador
Inmaculada Concepción Almor-Palacios
José M. Millán
Gema García-García
author_sort Belén García-Bohórquez
collection DOAJ
description Abstract Background Ciliopathies are characterized by the dysfunction of cilia, being inherited retinal dystrophies (IRDs) included in sensory ciliopathies. Besides, oro-facial-digital syndrome (OFD) is caused by mutations in ciliary genes, leading to dysmorphic features. Mutations in TBC1D32 were associated to retinal dystrophy and OFD, defining this form as OFD-IX. Results A clinical exome analysis performed on a patient presenting with OFD-IX and sensorineural hearing loss (SNHL) identified two variants in TBC1D32, one of which affects splicing, with its impact validated using a minigene assay. Conclusions These results suggest that SNHL may represent a new clinical feature associated with this gene.
format Article
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institution Kabale University
issn 1479-7364
language English
publishDate 2025-05-01
publisher BMC
record_format Article
series Human Genomics
spelling doaj-art-106692b1d6e1442e81a1f4a1493f57082025-08-20T03:52:19ZengBMCHuman Genomics1479-73642025-05-011911610.1186/s40246-025-00759-0Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndromeBelén García-Bohórquez0Purificación Marín-Reina1Elena Aller2Pilar Barberán-Martínez3Miguel Armengot4Roberto Llorens-Salvador5Inmaculada Concepción Almor-Palacios6José M. Millán7Gema García-García8Molecular, Cellular and Genomics Biomedicine, Health Research Institute La FeDivision of Neonatology, University and Polytechnic Hospital La FeMolecular, Cellular and Genomics Biomedicine, Health Research Institute La FeMolecular, Cellular and Genomics Biomedicine, Health Research Institute La FeDepartment of Otorhinolaryngology, and Primary Ciliary Dyskinesia Unit, University and Polytechnic Hospital La FeClinical Imaging Area, University and Polytechnic Hospital La FeDepartment of Ophthalmology, University and Polytechnic La Fe HospitalMolecular, Cellular and Genomics Biomedicine, Health Research Institute La FeMolecular, Cellular and Genomics Biomedicine, Health Research Institute La FeAbstract Background Ciliopathies are characterized by the dysfunction of cilia, being inherited retinal dystrophies (IRDs) included in sensory ciliopathies. Besides, oro-facial-digital syndrome (OFD) is caused by mutations in ciliary genes, leading to dysmorphic features. Mutations in TBC1D32 were associated to retinal dystrophy and OFD, defining this form as OFD-IX. Results A clinical exome analysis performed on a patient presenting with OFD-IX and sensorineural hearing loss (SNHL) identified two variants in TBC1D32, one of which affects splicing, with its impact validated using a minigene assay. Conclusions These results suggest that SNHL may represent a new clinical feature associated with this gene.https://doi.org/10.1186/s40246-025-00759-0TBC1D32Retinitis pigmentosaHearing lossMinigene assayExpanding clinical spectrum
spellingShingle Belén García-Bohórquez
Purificación Marín-Reina
Elena Aller
Pilar Barberán-Martínez
Miguel Armengot
Roberto Llorens-Salvador
Inmaculada Concepción Almor-Palacios
José M. Millán
Gema García-García
Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndrome
Human Genomics
TBC1D32
Retinitis pigmentosa
Hearing loss
Minigene assay
Expanding clinical spectrum
title Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndrome
title_full Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndrome
title_fullStr Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndrome
title_full_unstemmed Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndrome
title_short Two novel mutations in TBC1D32 add complexity to the oro-facial-digital syndrome
title_sort two novel mutations in tbc1d32 add complexity to the oro facial digital syndrome
topic TBC1D32
Retinitis pigmentosa
Hearing loss
Minigene assay
Expanding clinical spectrum
url https://doi.org/10.1186/s40246-025-00759-0
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