Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular Diseases

Human salusin-α and salusin-β are related peptides produced from prosalusin. Bolus injection of salusin-β into rats induces more profound hypotension and bradycardia than salusin-α. Central administration of salusin-β increases blood pressure via release of norepinephrine and arginine-vasopressin. C...

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Main Authors: Kengo Sato, Rena Watanabe, Fumiko Itoh, Masayoshi Shichiri, Takuya Watanabe
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:International Journal of Hypertension
Online Access:http://dx.doi.org/10.1155/2013/965140
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author Kengo Sato
Rena Watanabe
Fumiko Itoh
Masayoshi Shichiri
Takuya Watanabe
author_facet Kengo Sato
Rena Watanabe
Fumiko Itoh
Masayoshi Shichiri
Takuya Watanabe
author_sort Kengo Sato
collection DOAJ
description Human salusin-α and salusin-β are related peptides produced from prosalusin. Bolus injection of salusin-β into rats induces more profound hypotension and bradycardia than salusin-α. Central administration of salusin-β increases blood pressure via release of norepinephrine and arginine-vasopressin. Circulating levels of salusin-α and salusin-β are lower in patients with essential hypertension. Salusin-β exerts more potent mitogenic effects on human vascular smooth muscle cells (VSMCs) and fibroblasts than salusin-α. Salusin-β accelerates inflammatory responses in human endothelial cells and monocyte-endothelial adhesion. Human macrophage foam cell formation is stimulated by salusin-β but suppressed by salusin-α. Chronic salusin-β infusion into apolipoprotein E-deficient mice enhances atherosclerotic lesions; salusin-α infusion reduces lesions. Salusin-β is expressed in proliferative neointimal lesions of porcine coronary arteries after stenting. Salusin-α and salusin-β immunoreactivity have been detected in human coronary atherosclerotic plaques, with dominance of salusin-β in macrophage foam cells, VSMCs, and fibroblasts. Circulating salusin-β levels increase and salusin-α levels decrease in patients with coronary artery disease. These findings suggest that salusin-β and salusin-α may contribute to proatherogenesis and antiatherogenesis, respectively. Increased salusin-β and/or decreased salusin-α levels in circulating blood and vascular tissue are closely linked with atherosclerosis. Salusin-α and salusin-β could be candidate biomarkers and therapeutic targets for atherosclerotic cardiovascular diseases.
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spelling doaj-art-100eaf6cee024f16a2ed60b46c5505c82025-08-20T02:02:32ZengWileyInternational Journal of Hypertension2090-03842090-03922013-01-01201310.1155/2013/965140965140Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular DiseasesKengo Sato0Rena Watanabe1Fumiko Itoh2Masayoshi Shichiri3Takuya Watanabe4Laboratory of Cardiovascular Medicine, Tokyo University of Pharmacy and Life Sciences, 1432-1 Horinouchi, Hachioji, Tokyo 192-0392, JapanLaboratory of Cardiovascular Medicine, Tokyo University of Pharmacy and Life Sciences, 1432-1 Horinouchi, Hachioji, Tokyo 192-0392, JapanLaboratory of Cardiovascular Medicine, Tokyo University of Pharmacy and Life Sciences, 1432-1 Horinouchi, Hachioji, Tokyo 192-0392, JapanDepartment of Endocrinology, Diabetes and Metabolism, Kitasato University School of Medicine, 1-15-1 Kitasato, Minami-Ku, Sagamihara, Kanagawa 252-0374, JapanLaboratory of Cardiovascular Medicine, Tokyo University of Pharmacy and Life Sciences, 1432-1 Horinouchi, Hachioji, Tokyo 192-0392, JapanHuman salusin-α and salusin-β are related peptides produced from prosalusin. Bolus injection of salusin-β into rats induces more profound hypotension and bradycardia than salusin-α. Central administration of salusin-β increases blood pressure via release of norepinephrine and arginine-vasopressin. Circulating levels of salusin-α and salusin-β are lower in patients with essential hypertension. Salusin-β exerts more potent mitogenic effects on human vascular smooth muscle cells (VSMCs) and fibroblasts than salusin-α. Salusin-β accelerates inflammatory responses in human endothelial cells and monocyte-endothelial adhesion. Human macrophage foam cell formation is stimulated by salusin-β but suppressed by salusin-α. Chronic salusin-β infusion into apolipoprotein E-deficient mice enhances atherosclerotic lesions; salusin-α infusion reduces lesions. Salusin-β is expressed in proliferative neointimal lesions of porcine coronary arteries after stenting. Salusin-α and salusin-β immunoreactivity have been detected in human coronary atherosclerotic plaques, with dominance of salusin-β in macrophage foam cells, VSMCs, and fibroblasts. Circulating salusin-β levels increase and salusin-α levels decrease in patients with coronary artery disease. These findings suggest that salusin-β and salusin-α may contribute to proatherogenesis and antiatherogenesis, respectively. Increased salusin-β and/or decreased salusin-α levels in circulating blood and vascular tissue are closely linked with atherosclerosis. Salusin-α and salusin-β could be candidate biomarkers and therapeutic targets for atherosclerotic cardiovascular diseases.http://dx.doi.org/10.1155/2013/965140
spellingShingle Kengo Sato
Rena Watanabe
Fumiko Itoh
Masayoshi Shichiri
Takuya Watanabe
Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular Diseases
International Journal of Hypertension
title Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular Diseases
title_full Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular Diseases
title_fullStr Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular Diseases
title_full_unstemmed Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular Diseases
title_short Salusins: Potential Use as a Biomarker for Atherosclerotic Cardiovascular Diseases
title_sort salusins potential use as a biomarker for atherosclerotic cardiovascular diseases
url http://dx.doi.org/10.1155/2013/965140
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AT masayoshishichiri salusinspotentialuseasabiomarkerforatheroscleroticcardiovasculardiseases
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