A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin

Tumor heterogeneity contributes significantly to chemoresistance, a leading cause of treatment failure. To better personalize therapies, it is essential to develop tools capable of identifying and predicting intra- and inter-tumor heterogeneities. Biology-inspired mathematical models are capable of...

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Main Authors: Hugo J. M. Miniere, Ernesto A. B. F. Lima, Guillermo Lorenzo, David A. Hormuth II, Sophia Ty, Amy Brock, Thomas E. Yankeelov
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Cancer Biology & Therapy
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Online Access:https://www.tandfonline.com/doi/10.1080/15384047.2024.2321769
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author Hugo J. M. Miniere
Ernesto A. B. F. Lima
Guillermo Lorenzo
David A. Hormuth II
Sophia Ty
Amy Brock
Thomas E. Yankeelov
author_facet Hugo J. M. Miniere
Ernesto A. B. F. Lima
Guillermo Lorenzo
David A. Hormuth II
Sophia Ty
Amy Brock
Thomas E. Yankeelov
author_sort Hugo J. M. Miniere
collection DOAJ
description Tumor heterogeneity contributes significantly to chemoresistance, a leading cause of treatment failure. To better personalize therapies, it is essential to develop tools capable of identifying and predicting intra- and inter-tumor heterogeneities. Biology-inspired mathematical models are capable of attacking this problem, but tumor heterogeneity is often overlooked in in-vivo modeling studies, while phenotypic considerations capturing spatial dynamics are not typically included in in-vitro modeling studies. We present a data assimilation-prediction pipeline with a two-phenotype model that includes a spatiotemporal component to characterize and predict the evolution of in-vitro breast cancer cells and their heterogeneous response to chemotherapy. Our model assumes that the cells can be divided into two subpopulations: surviving cells unaffected by the treatment, and irreversibly damaged cells undergoing treatment-induced death. MCF7 breast cancer cells were previously cultivated in wells for up to 1000 hours, treated with various concentrations of doxorubicin and imaged with time-resolved microscopy to record spatiotemporally-resolved cell count data. Images were used to generate cell density maps. Treatment response predictions were initialized by a training set and updated by weekly measurements. Our mathematical model successfully calibrated the spatiotemporal cell growth dynamics, achieving median [range] concordance correlation coefficients of > .99 [.88, >.99] and .73 [.58, .85] across the whole well and individual pixels, respectively. Our proposed data assimilation-prediction approach achieved values of .97 [.44, >.99] and .69 [.35, .79] for the whole well and individual pixels, respectively. Thus, our model can capture and predict the spatiotemporal dynamics of MCF7 cells treated with doxorubicin in an in-vitro setting.
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spelling doaj-art-0e280a58802b47fcb80eb711f4c69ab32025-08-20T02:49:47ZengTaylor & Francis GroupCancer Biology & Therapy1538-40471555-85762024-12-0125110.1080/15384047.2024.2321769A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicinHugo J. M. Miniere0Ernesto A. B. F. Lima1Guillermo Lorenzo2David A. Hormuth II3Sophia Ty4Amy Brock5Thomas E. Yankeelov6Department of Biomedical Engineering, The University of Texas at Austin, Austin, USAOden Institute for Computational Engineering and Sciences, The University of Texas at Austin, Austin, USAOden Institute for Computational Engineering and Sciences, The University of Texas at Austin, Austin, USAOden Institute for Computational Engineering and Sciences, The University of Texas at Austin, Austin, USADepartment of Biomedical Engineering, The University of Texas at Austin, Austin, USADepartment of Biomedical Engineering, The University of Texas at Austin, Austin, USADepartment of Biomedical Engineering, The University of Texas at Austin, Austin, USATumor heterogeneity contributes significantly to chemoresistance, a leading cause of treatment failure. To better personalize therapies, it is essential to develop tools capable of identifying and predicting intra- and inter-tumor heterogeneities. Biology-inspired mathematical models are capable of attacking this problem, but tumor heterogeneity is often overlooked in in-vivo modeling studies, while phenotypic considerations capturing spatial dynamics are not typically included in in-vitro modeling studies. We present a data assimilation-prediction pipeline with a two-phenotype model that includes a spatiotemporal component to characterize and predict the evolution of in-vitro breast cancer cells and their heterogeneous response to chemotherapy. Our model assumes that the cells can be divided into two subpopulations: surviving cells unaffected by the treatment, and irreversibly damaged cells undergoing treatment-induced death. MCF7 breast cancer cells were previously cultivated in wells for up to 1000 hours, treated with various concentrations of doxorubicin and imaged with time-resolved microscopy to record spatiotemporally-resolved cell count data. Images were used to generate cell density maps. Treatment response predictions were initialized by a training set and updated by weekly measurements. Our mathematical model successfully calibrated the spatiotemporal cell growth dynamics, achieving median [range] concordance correlation coefficients of > .99 [.88, >.99] and .73 [.58, .85] across the whole well and individual pixels, respectively. Our proposed data assimilation-prediction approach achieved values of .97 [.44, >.99] and .69 [.35, .79] for the whole well and individual pixels, respectively. Thus, our model can capture and predict the spatiotemporal dynamics of MCF7 cells treated with doxorubicin in an in-vitro setting.https://www.tandfonline.com/doi/10.1080/15384047.2024.2321769Time-resolved microscopydata assimilationmechanism-based modelingmathematical oncology
spellingShingle Hugo J. M. Miniere
Ernesto A. B. F. Lima
Guillermo Lorenzo
David A. Hormuth II
Sophia Ty
Amy Brock
Thomas E. Yankeelov
A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin
Cancer Biology & Therapy
Time-resolved microscopy
data assimilation
mechanism-based modeling
mathematical oncology
title A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin
title_full A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin
title_fullStr A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin
title_full_unstemmed A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin
title_short A mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin
title_sort mathematical model for predicting the spatiotemporal response of breast cancer cells treated with doxorubicin
topic Time-resolved microscopy
data assimilation
mechanism-based modeling
mathematical oncology
url https://www.tandfonline.com/doi/10.1080/15384047.2024.2321769
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