The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH protein

Abstract Triple-negative breast cancer (TNBC) is an aggressive subtype of invasive breast cancer characterized by limited treatment options and a poor prognosis. While ferroptosis, an iron-dependent form of regulated cell death, plays a role in tumor suppression, its specific molecular mechanisms in...

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Main Authors: Li Yang, Xiaoqin An, Shangzhu Yang, Xiaowen Lin, Ziyuan Chen, Qian Xue, Xi Chen, Yuan Wang, Ding Yan, Shirui Chen, Yuqing Fan, Daolin Tang, Wenfeng Yu, Jinbao Liu, Xin Chen
Format: Article
Language:English
Published: Nature Publishing Group 2025-07-01
Series:Cell Death and Disease
Online Access:https://doi.org/10.1038/s41419-025-07895-4
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author Li Yang
Xiaoqin An
Shangzhu Yang
Xiaowen Lin
Ziyuan Chen
Qian Xue
Xi Chen
Yuan Wang
Ding Yan
Shirui Chen
Yuqing Fan
Daolin Tang
Wenfeng Yu
Jinbao Liu
Xin Chen
author_facet Li Yang
Xiaoqin An
Shangzhu Yang
Xiaowen Lin
Ziyuan Chen
Qian Xue
Xi Chen
Yuan Wang
Ding Yan
Shirui Chen
Yuqing Fan
Daolin Tang
Wenfeng Yu
Jinbao Liu
Xin Chen
author_sort Li Yang
collection DOAJ
description Abstract Triple-negative breast cancer (TNBC) is an aggressive subtype of invasive breast cancer characterized by limited treatment options and a poor prognosis. While ferroptosis, an iron-dependent form of regulated cell death, plays a role in tumor suppression, its specific molecular mechanisms in TNBC remain largely unexplored. In this study, we identify deubiquitinase USP24 as the most significantly altered enzyme among key deubiquitinating enzymes during ferroptosis in human TNBC cells. Silencing USP24 enhances ferroptosis-mediated tumor suppression in TNBC cells. Mechanistically, USP24 interacts directly with dihydroorotate dehydrogenase (DHODH) and deubiquitinates it, a process critical for maintaining coenzyme Q reduction and protecting cells from lipid peroxidation. Consistently, pharmacological inhibition of USP24 synergizes strongly with ferroptosis inducers in both in vitro and in vivo models via a DHODH-dependent pathway. These findings highlight USP24 as a potential therapeutic target to enhance ferroptosis sensitivity in TNBC.
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institution Kabale University
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language English
publishDate 2025-07-01
publisher Nature Publishing Group
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series Cell Death and Disease
spelling doaj-art-0dcf1a8a50c74291942c4b8f720641882025-08-20T04:02:41ZengNature Publishing GroupCell Death and Disease2041-48892025-07-0116111510.1038/s41419-025-07895-4The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH proteinLi Yang0Xiaoqin An1Shangzhu Yang2Xiaowen Lin3Ziyuan Chen4Qian Xue5Xi Chen6Yuan Wang7Ding Yan8Shirui Chen9Yuqing Fan10Daolin Tang11Wenfeng Yu12Jinbao Liu13Xin Chen14Department of Physiology, School of Basic Medical Sciences, Guizhou Medical UniversityDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical UniversityDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical UniversityKey Laboratory of Biological Targeting Diagnosis, Therapy and Rehabilitation of Guangdong Higher Education Institutes, The Fifth Affiliated Hospital, Guangzhou Medical UniversityKey Laboratory of Biological Targeting Diagnosis, Therapy and Rehabilitation of Guangdong Higher Education Institutes, The Fifth Affiliated Hospital, Guangzhou Medical UniversityGuangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Disease, School of Basic Medical Sciences, Guangzhou Medical UniversityGuangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Disease, School of Basic Medical Sciences, Guangzhou Medical UniversityKey Laboratory of Biological Targeting Diagnosis, Therapy and Rehabilitation of Guangdong Higher Education Institutes, The Fifth Affiliated Hospital, Guangzhou Medical UniversityKey Laboratory of Biological Targeting Diagnosis, Therapy and Rehabilitation of Guangdong Higher Education Institutes, The Fifth Affiliated Hospital, Guangzhou Medical UniversityDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical UniversityDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical UniversityDepartment of Surgery, UT Southwestern Medical CenterDepartment of Physiology, School of Basic Medical Sciences, Guizhou Medical UniversityGuangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Disease, School of Basic Medical Sciences, Guangzhou Medical UniversityKey Laboratory of Biological Targeting Diagnosis, Therapy and Rehabilitation of Guangdong Higher Education Institutes, The Fifth Affiliated Hospital, Guangzhou Medical UniversityAbstract Triple-negative breast cancer (TNBC) is an aggressive subtype of invasive breast cancer characterized by limited treatment options and a poor prognosis. While ferroptosis, an iron-dependent form of regulated cell death, plays a role in tumor suppression, its specific molecular mechanisms in TNBC remain largely unexplored. In this study, we identify deubiquitinase USP24 as the most significantly altered enzyme among key deubiquitinating enzymes during ferroptosis in human TNBC cells. Silencing USP24 enhances ferroptosis-mediated tumor suppression in TNBC cells. Mechanistically, USP24 interacts directly with dihydroorotate dehydrogenase (DHODH) and deubiquitinates it, a process critical for maintaining coenzyme Q reduction and protecting cells from lipid peroxidation. Consistently, pharmacological inhibition of USP24 synergizes strongly with ferroptosis inducers in both in vitro and in vivo models via a DHODH-dependent pathway. These findings highlight USP24 as a potential therapeutic target to enhance ferroptosis sensitivity in TNBC.https://doi.org/10.1038/s41419-025-07895-4
spellingShingle Li Yang
Xiaoqin An
Shangzhu Yang
Xiaowen Lin
Ziyuan Chen
Qian Xue
Xi Chen
Yuan Wang
Ding Yan
Shirui Chen
Yuqing Fan
Daolin Tang
Wenfeng Yu
Jinbao Liu
Xin Chen
The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH protein
Cell Death and Disease
title The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH protein
title_full The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH protein
title_fullStr The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH protein
title_full_unstemmed The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH protein
title_short The deubiquitinase USP24 suppresses ferroptosis in triple-negative breast cancer by stabilizing DHODH protein
title_sort deubiquitinase usp24 suppresses ferroptosis in triple negative breast cancer by stabilizing dhodh protein
url https://doi.org/10.1038/s41419-025-07895-4
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