Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity

Abstract This cross-sectional study explores the impact of FTO gene single nucleotide polymorphisms (SNPs) rs9939609 and rs1421085 on dietary habits contributing to obesity risk in Thai adults. The study enrolled 384 participants from Bangkok, categorized as non-obese (BMI < 25 kg/m2) or obese (B...

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Main Authors: Sakawrut Poosri, Usa Boonyuen, Chaowanee Chupeerach, Ngamphol Soonthornworasiri, Karunee Kwanbunjan, Pattaneeya Prangthip
Format: Article
Language:English
Published: Nature Portfolio 2024-10-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-024-77004-6
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author Sakawrut Poosri
Usa Boonyuen
Chaowanee Chupeerach
Ngamphol Soonthornworasiri
Karunee Kwanbunjan
Pattaneeya Prangthip
author_facet Sakawrut Poosri
Usa Boonyuen
Chaowanee Chupeerach
Ngamphol Soonthornworasiri
Karunee Kwanbunjan
Pattaneeya Prangthip
author_sort Sakawrut Poosri
collection DOAJ
description Abstract This cross-sectional study explores the impact of FTO gene single nucleotide polymorphisms (SNPs) rs9939609 and rs1421085 on dietary habits contributing to obesity risk in Thai adults. The study enrolled 384 participants from Bangkok, categorized as non-obese (BMI < 25 kg/m2) or obese (BMI ≥ 25 kg/m2) based on WHO Asia Pacific Guidelines. Genotyping for FTO variants was performed using DNA from blood samples. While both SNPs adhered to Hardy–Weinberg equilibrium, the association between risk alleles and anthropometric measurements was not statistically significant. However, risk allele carriers showed significantly higher intakes of sugar and saturated fat compared to homozygous dominant individuals. In the obese group, the odds ratio for high-sugar intake was 2.22 (95% CI 1.13–4.37, p = 0.021) for rs9939609 risk allele carriers. For high-saturated fat intake, the odds ratio was 1.86 (95% CI 1.02–3.40, p = 0.041). Similar associations were observed for rs1421085. Risk allele carriers also exhibited significantly higher leptin levels (p < 0.043) and a positive correlation with myeloperoxidase levels (p < 0.038). These findings highlight the complex relationship between FTO risk alleles, increased consumption of sugar and saturated fat, and obesity-related parameters. The insights emphasize the importance of considering both genetic and dietary factors in obesity prevention strategies.
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spelling doaj-art-0cb4bc1ebd3c41828483fcb7736f480a2025-08-20T02:11:22ZengNature PortfolioScientific Reports2045-23222024-10-0114111510.1038/s41598-024-77004-6Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesitySakawrut Poosri0Usa Boonyuen1Chaowanee Chupeerach2Ngamphol Soonthornworasiri3Karunee Kwanbunjan4Pattaneeya Prangthip5Department of Tropical Nutrition and Food Science, Faculty of Tropical Medicine, Mahidol UniversityDepartment of Molecular Tropical Medicine and Genetics, Faculty of Tropical Medicine, Mahidol UniversityFood and Nutrition Academic and Research Cluster, Institute of Nutrition, Mahidol UniversityDepartment of Tropical Hygiene, Faculty of Tropical Medicine, Mahidol UniversityDepartment of Tropical Nutrition and Food Science, Faculty of Tropical Medicine, Mahidol UniversityDepartment of Tropical Nutrition and Food Science, Faculty of Tropical Medicine, Mahidol UniversityAbstract This cross-sectional study explores the impact of FTO gene single nucleotide polymorphisms (SNPs) rs9939609 and rs1421085 on dietary habits contributing to obesity risk in Thai adults. The study enrolled 384 participants from Bangkok, categorized as non-obese (BMI < 25 kg/m2) or obese (BMI ≥ 25 kg/m2) based on WHO Asia Pacific Guidelines. Genotyping for FTO variants was performed using DNA from blood samples. While both SNPs adhered to Hardy–Weinberg equilibrium, the association between risk alleles and anthropometric measurements was not statistically significant. However, risk allele carriers showed significantly higher intakes of sugar and saturated fat compared to homozygous dominant individuals. In the obese group, the odds ratio for high-sugar intake was 2.22 (95% CI 1.13–4.37, p = 0.021) for rs9939609 risk allele carriers. For high-saturated fat intake, the odds ratio was 1.86 (95% CI 1.02–3.40, p = 0.041). Similar associations were observed for rs1421085. Risk allele carriers also exhibited significantly higher leptin levels (p < 0.043) and a positive correlation with myeloperoxidase levels (p < 0.038). These findings highlight the complex relationship between FTO risk alleles, increased consumption of sugar and saturated fat, and obesity-related parameters. The insights emphasize the importance of considering both genetic and dietary factors in obesity prevention strategies.https://doi.org/10.1038/s41598-024-77004-6ObesityFTO geneSingle nucleotide polymorphismsDietary intakeGenetic riskHuman genetics
spellingShingle Sakawrut Poosri
Usa Boonyuen
Chaowanee Chupeerach
Ngamphol Soonthornworasiri
Karunee Kwanbunjan
Pattaneeya Prangthip
Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity
Scientific Reports
Obesity
FTO gene
Single nucleotide polymorphisms
Dietary intake
Genetic risk
Human genetics
title Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity
title_full Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity
title_fullStr Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity
title_full_unstemmed Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity
title_short Association of FTO variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity
title_sort association of fto variants rs9939609 and rs1421085 with elevated sugar and fat consumption in adult obesity
topic Obesity
FTO gene
Single nucleotide polymorphisms
Dietary intake
Genetic risk
Human genetics
url https://doi.org/10.1038/s41598-024-77004-6
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