Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016

Abstract Organophosphate esters (OPEs) exposure has potentially harmful effects on human health. However, the evidence between OPEs and hyperuricemia is insufficient. We aimed to assess the association between OPEs metabolites and the prevalence of hyperuricemia. Multivariable logistic regression, w...

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Main Authors: Qiong Huang, Wenbin Nan, Siqi Li, Baimei He, Xu Cai, Zhenyu Peng, Chenlu Wu
Format: Article
Language:English
Published: Nature Portfolio 2025-04-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-96423-7
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author Qiong Huang
Wenbin Nan
Siqi Li
Baimei He
Xu Cai
Zhenyu Peng
Chenlu Wu
author_facet Qiong Huang
Wenbin Nan
Siqi Li
Baimei He
Xu Cai
Zhenyu Peng
Chenlu Wu
author_sort Qiong Huang
collection DOAJ
description Abstract Organophosphate esters (OPEs) exposure has potentially harmful effects on human health. However, the evidence between OPEs and hyperuricemia is insufficient. We aimed to assess the association between OPEs metabolites and the prevalence of hyperuricemia. Multivariable logistic regression, weighted quantile regression (WQS) model, and Bayesian kernel machine regression (BKMR) models were used to investigate the association of OPEs metabolites with the risk of hyperuricemia. Mediation analysis was conducted to assess whether inflammation mediated the effects of OPEs on the prevalence of hyperuricemia. The multivariable logistics regression indicated that bis (1,3-dichloro-2-propyl) phosphate (BDCPP) and bis-2-chloroethyl phosphate (BCEP) were positively correlated with the risk of hyperuricemia. In WQS and BKMR analyses, OPEs mixtures presented a positive association with the risk of hyperuricemia, with BDCPP being the primary contributor. C-reactive protein (CRP) and monocytes were found to mediate the association between BDCPP and the risk of hyperuricemia prevalence, with 8.46% and 3.97% of the mediated proportion, respectively. Our study revealed that OPEs mixtures were positively correlated with the prevalence of hyperuricemia, with BDCPP identified as the most significant contributor. Inflammation was a potential mechanism mediating the effect of BDCPP exposure on the risk of hyperuricemia.
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spelling doaj-art-0afe85ce7c504ceaa75acdb2d5968bc92025-08-20T02:17:09ZengNature PortfolioScientific Reports2045-23222025-04-0115111110.1038/s41598-025-96423-7Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016Qiong Huang0Wenbin Nan1Siqi Li2Baimei He3Xu Cai4Zhenyu Peng5Chenlu Wu6Department of Metabolism and Endocrinology, Second Xiangya Hospital, Central South UniversityDepartment of Emergency Medicine, Second Xiangya Hospital, Central South UniversityDepartment of Geriatric Respiratory and Critical Care Medicine, Xiangya Hospital, Central South UniversityDepartment of Geriatric Respiratory and Critical Care Medicine, Xiangya Hospital, Central South UniversityDepartment of Emergency Medicine, Second Xiangya Hospital, Central South UniversityDepartment of Emergency Medicine, Second Xiangya Hospital, Central South UniversityDepartment of Cardiology, Second Xiangya Hospital, Central South UniversityAbstract Organophosphate esters (OPEs) exposure has potentially harmful effects on human health. However, the evidence between OPEs and hyperuricemia is insufficient. We aimed to assess the association between OPEs metabolites and the prevalence of hyperuricemia. Multivariable logistic regression, weighted quantile regression (WQS) model, and Bayesian kernel machine regression (BKMR) models were used to investigate the association of OPEs metabolites with the risk of hyperuricemia. Mediation analysis was conducted to assess whether inflammation mediated the effects of OPEs on the prevalence of hyperuricemia. The multivariable logistics regression indicated that bis (1,3-dichloro-2-propyl) phosphate (BDCPP) and bis-2-chloroethyl phosphate (BCEP) were positively correlated with the risk of hyperuricemia. In WQS and BKMR analyses, OPEs mixtures presented a positive association with the risk of hyperuricemia, with BDCPP being the primary contributor. C-reactive protein (CRP) and monocytes were found to mediate the association between BDCPP and the risk of hyperuricemia prevalence, with 8.46% and 3.97% of the mediated proportion, respectively. Our study revealed that OPEs mixtures were positively correlated with the prevalence of hyperuricemia, with BDCPP identified as the most significant contributor. Inflammation was a potential mechanism mediating the effect of BDCPP exposure on the risk of hyperuricemia.https://doi.org/10.1038/s41598-025-96423-7HyperuricemiaOrganophosphate estersInflammationNHANESOPEs
spellingShingle Qiong Huang
Wenbin Nan
Siqi Li
Baimei He
Xu Cai
Zhenyu Peng
Chenlu Wu
Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016
Scientific Reports
Hyperuricemia
Organophosphate esters
Inflammation
NHANES
OPEs
title Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016
title_full Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016
title_fullStr Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016
title_full_unstemmed Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016
title_short Association between organophosphate esters exposure and the prevalence of hyperuricemia in US adults from NHANES 2011–2016
title_sort association between organophosphate esters exposure and the prevalence of hyperuricemia in us adults from nhanes 2011 2016
topic Hyperuricemia
Organophosphate esters
Inflammation
NHANES
OPEs
url https://doi.org/10.1038/s41598-025-96423-7
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