Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretion

Background Platinum is a commonly used drug for ovarian cancer (OvCa) treatment, but drug resistance limits its clinical application. This study intended to delineate the effects of adipocytes on platinum resistance in OvCa.Methods OvCa cells were maintained in the adipocyte-conditioned medium. Cell...

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Main Authors: Jiong Ma, Junyan Li, Xuejun Chen, Yanyan Ma
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Adipocyte
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Online Access:https://www.tandfonline.com/doi/10.1080/21623945.2023.2282566
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author Jiong Ma
Junyan Li
Xuejun Chen
Yanyan Ma
author_facet Jiong Ma
Junyan Li
Xuejun Chen
Yanyan Ma
author_sort Jiong Ma
collection DOAJ
description Background Platinum is a commonly used drug for ovarian cancer (OvCa) treatment, but drug resistance limits its clinical application. This study intended to delineate the effects of adipocytes on platinum resistance in OvCa.Methods OvCa cells were maintained in the adipocyte-conditioned medium. Cell viability and apoptosis were detected by CCK-8 and flow cytometry, separately. Proliferation and apoptosis-related protein expression were assayed by western blot. The IC50 values of cisplatin and carboplatin were determined using CCK-8. IGF1 secretion and expression were assayed via ELISA and western blot, respectively. A xenograft model was established, and pathological changes were detected by H&E staining. Proliferation and apoptosis-associated protein expression was assessed via IHC.Results Adipocytes promoted the viability and repressed cell apoptosis in OvCa, as well as enhancing platinum resistance, while the addition of IGF-1 R inhibitor reversed the effects of adipocytes on proliferation, apoptosis, and drug resistance of OvCa cells. Treatment with different concentrations of Ojeok-san (OJS) inhibited the adipocyte-induced platinum resistance in OvCa cells by suppressing IGF1. The combined treatment of OJS and cisplatin significantly inhibited tumour growth in vivo with good mouse tolerance.Conclusion In summary, OJS inhibited OvCa proliferation and platinum resistance by suppressing adipocyte paracrine IGF1 secretion.
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spelling doaj-art-0ae3bd5423bc406aa477b6c5a8567e952025-08-20T02:30:41ZengTaylor & Francis GroupAdipocyte2162-39452162-397X2024-12-0113110.1080/21623945.2023.2282566Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretionJiong Ma0Junyan Li1Xuejun Chen2Yanyan Ma3Department of Gynecology, The Second Affiliated Hospital, Zhejiang University, School of Medicine, Hangzhou, ChinaDepartment of Gynecology, The Second Affiliated Hospital, Zhejiang University, School of Medicine, Hangzhou, ChinaDepartment of Gynecology, The Second Affiliated Hospital, Zhejiang University, School of Medicine, Hangzhou, ChinaDepartment of Gynecology, The Second Affiliated Hospital, Zhejiang University, School of Medicine, Hangzhou, ChinaBackground Platinum is a commonly used drug for ovarian cancer (OvCa) treatment, but drug resistance limits its clinical application. This study intended to delineate the effects of adipocytes on platinum resistance in OvCa.Methods OvCa cells were maintained in the adipocyte-conditioned medium. Cell viability and apoptosis were detected by CCK-8 and flow cytometry, separately. Proliferation and apoptosis-related protein expression were assayed by western blot. The IC50 values of cisplatin and carboplatin were determined using CCK-8. IGF1 secretion and expression were assayed via ELISA and western blot, respectively. A xenograft model was established, and pathological changes were detected by H&E staining. Proliferation and apoptosis-associated protein expression was assessed via IHC.Results Adipocytes promoted the viability and repressed cell apoptosis in OvCa, as well as enhancing platinum resistance, while the addition of IGF-1 R inhibitor reversed the effects of adipocytes on proliferation, apoptosis, and drug resistance of OvCa cells. Treatment with different concentrations of Ojeok-san (OJS) inhibited the adipocyte-induced platinum resistance in OvCa cells by suppressing IGF1. The combined treatment of OJS and cisplatin significantly inhibited tumour growth in vivo with good mouse tolerance.Conclusion In summary, OJS inhibited OvCa proliferation and platinum resistance by suppressing adipocyte paracrine IGF1 secretion.https://www.tandfonline.com/doi/10.1080/21623945.2023.2282566Ovarian canceradipocyteIGF1platinumOJS
spellingShingle Jiong Ma
Junyan Li
Xuejun Chen
Yanyan Ma
Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretion
Adipocyte
Ovarian cancer
adipocyte
IGF1
platinum
OJS
title Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretion
title_full Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretion
title_fullStr Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretion
title_full_unstemmed Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretion
title_short Ojeok-san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine IGF1 secretion
title_sort ojeok san enhances platinum sensitivity in ovarian cancer by regulating adipocyte paracrine igf1 secretion
topic Ovarian cancer
adipocyte
IGF1
platinum
OJS
url https://www.tandfonline.com/doi/10.1080/21623945.2023.2282566
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AT junyanli ojeoksanenhancesplatinumsensitivityinovariancancerbyregulatingadipocyteparacrineigf1secretion
AT xuejunchen ojeoksanenhancesplatinumsensitivityinovariancancerbyregulatingadipocyteparacrineigf1secretion
AT yanyanma ojeoksanenhancesplatinumsensitivityinovariancancerbyregulatingadipocyteparacrineigf1secretion