PGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1

Abstract Abnormal expression of PGRP-S plays an important role in the development and progression of hepatocellular carcinoma (HCC), although the mechanism has remained elusive. In this study, we aimed to investigate the biological function and potential mechanism of PGRP-S in HCC. We found that PGR...

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Main Authors: Huifang Zhu, Shuang Feng, Sizhen Lv, Di Zhao, Xuanhe Chang, Shangqian Fan, Xia Wang, Xinlai Qian
Format: Article
Language:English
Published: Nature Portfolio 2025-08-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-12160-x
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author Huifang Zhu
Shuang Feng
Sizhen Lv
Di Zhao
Xuanhe Chang
Shangqian Fan
Xia Wang
Xinlai Qian
author_facet Huifang Zhu
Shuang Feng
Sizhen Lv
Di Zhao
Xuanhe Chang
Shangqian Fan
Xia Wang
Xinlai Qian
author_sort Huifang Zhu
collection DOAJ
description Abstract Abnormal expression of PGRP-S plays an important role in the development and progression of hepatocellular carcinoma (HCC), although the mechanism has remained elusive. In this study, we aimed to investigate the biological function and potential mechanism of PGRP-S in HCC. We found that PGRP-S was upregulated in HCC tissues compared to adjacent tissues. The increased expression of PGRP-S correlated with the differentiation and lymph node metastasis of HCC. PGRP-S obviously promoted HCC cell proliferation, migration and invasion in vitro. In vivo nude mouse models showed that PGRP-S enhanced tumor growth and pulmonary metastasis. Mechanistically, PGRP-S interacted with TTC1 and activated MAPK/ERK pathway, leading to HCC cell proliferation, migration and invasion. Therfore, our conclusion is PGRP-S promotes HCC cell proliferation, migration and invasion by activating MAPK/ERK pathway, which provides a new potential strategy for HCC treatment by targeting PGRP-S-TTC1 axis.
format Article
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institution Kabale University
issn 2045-2322
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publishDate 2025-08-01
publisher Nature Portfolio
record_format Article
series Scientific Reports
spelling doaj-art-06f3343008fa4ccca1609c398d0aa7852025-08-20T03:43:15ZengNature PortfolioScientific Reports2045-23222025-08-0115111610.1038/s41598-025-12160-xPGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1Huifang Zhu0Shuang Feng1Sizhen Lv2Di Zhao3Xuanhe Chang4Shangqian Fan5Xia Wang6Xinlai Qian7Department of Pathology, The Third Affiliated Hospital of Xinxiang Medical University, School of Basic Medical Sciences, Xinxiang Medical UniversityDepartment of Pathology, The Third Affiliated Hospital of Xinxiang Medical University, School of Basic Medical Sciences, Xinxiang Medical UniversityDepartment of Pathology, The Third Affiliated Hospital of Xinxiang Medical University, School of Basic Medical Sciences, Xinxiang Medical UniversityDepartment of Pathology, The Third Affiliated Hospital of Xinxiang Medical University, School of Basic Medical Sciences, Xinxiang Medical UniversityThe First Bethune Clinical Medical College of Ji Lin UniversityDepartment of Pathology, The Third Affiliated Hospital of Xinxiang Medical University, School of Basic Medical Sciences, Xinxiang Medical UniversityDepartment of Pathology, The Third Affiliated Hospital of Xinxiang Medical University, School of Basic Medical Sciences, Xinxiang Medical UniversityDepartment of Pathology, The Third Affiliated Hospital of Xinxiang Medical University, School of Basic Medical Sciences, Xinxiang Medical UniversityAbstract Abnormal expression of PGRP-S plays an important role in the development and progression of hepatocellular carcinoma (HCC), although the mechanism has remained elusive. In this study, we aimed to investigate the biological function and potential mechanism of PGRP-S in HCC. We found that PGRP-S was upregulated in HCC tissues compared to adjacent tissues. The increased expression of PGRP-S correlated with the differentiation and lymph node metastasis of HCC. PGRP-S obviously promoted HCC cell proliferation, migration and invasion in vitro. In vivo nude mouse models showed that PGRP-S enhanced tumor growth and pulmonary metastasis. Mechanistically, PGRP-S interacted with TTC1 and activated MAPK/ERK pathway, leading to HCC cell proliferation, migration and invasion. Therfore, our conclusion is PGRP-S promotes HCC cell proliferation, migration and invasion by activating MAPK/ERK pathway, which provides a new potential strategy for HCC treatment by targeting PGRP-S-TTC1 axis.https://doi.org/10.1038/s41598-025-12160-xPGRP-SHepatocellular carcinomaTTC1ProliferationMigration
spellingShingle Huifang Zhu
Shuang Feng
Sizhen Lv
Di Zhao
Xuanhe Chang
Shangqian Fan
Xia Wang
Xinlai Qian
PGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1
Scientific Reports
PGRP-S
Hepatocellular carcinoma
TTC1
Proliferation
Migration
title PGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1
title_full PGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1
title_fullStr PGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1
title_full_unstemmed PGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1
title_short PGRP-S promotes hepatocellular carcinoma progression via MAPK/ERK pathway by interaction with TTC1
title_sort pgrp s promotes hepatocellular carcinoma progression via mapk erk pathway by interaction with ttc1
topic PGRP-S
Hepatocellular carcinoma
TTC1
Proliferation
Migration
url https://doi.org/10.1038/s41598-025-12160-x
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