PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated Kinase

Proprotein convertase subtilisin/kexin type 9 can mediate the intracellular lysosomal degradation of the low-density lipoprotein receptor protein in hepatocytes and decrease the liver’s ability to scavenge low-density lipoprotein cholesterol from circulation, resulting in high levels of cholesterol...

Full description

Saved in:
Bibliographic Details
Main Authors: Lei Huang, Ying Cheng, Yulian Mu, Kui Li
Format: Article
Language:English
Published: Wiley 2023-01-01
Series:Analytical Cellular Pathology
Online Access:http://dx.doi.org/10.1155/2023/6985808
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849305626817069056
author Lei Huang
Ying Cheng
Yulian Mu
Kui Li
author_facet Lei Huang
Ying Cheng
Yulian Mu
Kui Li
author_sort Lei Huang
collection DOAJ
description Proprotein convertase subtilisin/kexin type 9 can mediate the intracellular lysosomal degradation of the low-density lipoprotein receptor protein in hepatocytes and decrease the liver’s ability to scavenge low-density lipoprotein cholesterol from circulation, resulting in high levels of cholesterol in the circulatory system. Current studies have primarily focused on the relationship between PCSK9 and blood lipid metabolism; however, the biological function of PCSK9 in hepatocytes is rarely addressed. In this study, we evaluate its effects in the human hepatoma carcinoma cell line HepG2, including proliferation, migration, and free cholesterol transport. PCSK9-D374Y is a gain-of-function mutation that does not affect proliferation but significantly suppresses the migration and cholesterol efflux capacity of these cells. The suppression of the transmembrane outflow of intracellular-free cholesterol regulates small G proteins and the suppression of extracellular signal-regulated kinase. In summary, PCSK9-D374Y affects hepatocyte features, including their migration and free cholesterol transport capabilities.
format Article
id doaj-art-0693d56e2b2a47f4b3f1b9b186c217d2
institution Kabale University
issn 2210-7185
language English
publishDate 2023-01-01
publisher Wiley
record_format Article
series Analytical Cellular Pathology
spelling doaj-art-0693d56e2b2a47f4b3f1b9b186c217d22025-08-20T03:55:23ZengWileyAnalytical Cellular Pathology2210-71852023-01-01202310.1155/2023/6985808PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated KinaseLei Huang0Ying Cheng1Yulian Mu2Kui Li3Shenzhen BranchShenzhen BranchState Key Laboratory of Animal NutritionShenzhen BranchProprotein convertase subtilisin/kexin type 9 can mediate the intracellular lysosomal degradation of the low-density lipoprotein receptor protein in hepatocytes and decrease the liver’s ability to scavenge low-density lipoprotein cholesterol from circulation, resulting in high levels of cholesterol in the circulatory system. Current studies have primarily focused on the relationship between PCSK9 and blood lipid metabolism; however, the biological function of PCSK9 in hepatocytes is rarely addressed. In this study, we evaluate its effects in the human hepatoma carcinoma cell line HepG2, including proliferation, migration, and free cholesterol transport. PCSK9-D374Y is a gain-of-function mutation that does not affect proliferation but significantly suppresses the migration and cholesterol efflux capacity of these cells. The suppression of the transmembrane outflow of intracellular-free cholesterol regulates small G proteins and the suppression of extracellular signal-regulated kinase. In summary, PCSK9-D374Y affects hepatocyte features, including their migration and free cholesterol transport capabilities.http://dx.doi.org/10.1155/2023/6985808
spellingShingle Lei Huang
Ying Cheng
Yulian Mu
Kui Li
PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated Kinase
Analytical Cellular Pathology
title PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated Kinase
title_full PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated Kinase
title_fullStr PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated Kinase
title_full_unstemmed PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated Kinase
title_short PCSK9-D374Y Suppresses Hepatocyte Migration through Downregulating Free Cholesterol Efflux Rate and Activity of Extracellular Signal-Regulated Kinase
title_sort pcsk9 d374y suppresses hepatocyte migration through downregulating free cholesterol efflux rate and activity of extracellular signal regulated kinase
url http://dx.doi.org/10.1155/2023/6985808
work_keys_str_mv AT leihuang pcsk9d374ysuppresseshepatocytemigrationthroughdownregulatingfreecholesteroleffluxrateandactivityofextracellularsignalregulatedkinase
AT yingcheng pcsk9d374ysuppresseshepatocytemigrationthroughdownregulatingfreecholesteroleffluxrateandactivityofextracellularsignalregulatedkinase
AT yulianmu pcsk9d374ysuppresseshepatocytemigrationthroughdownregulatingfreecholesteroleffluxrateandactivityofextracellularsignalregulatedkinase
AT kuili pcsk9d374ysuppresseshepatocytemigrationthroughdownregulatingfreecholesteroleffluxrateandactivityofextracellularsignalregulatedkinase