Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipients

Background: Donor-derived cell-free DNA (dd-cfDNA) is a valuable biomarker for allograft injury, but its association with various histopathologic injury patterns as specified in the Lung Allograft Standardized Histological Analysis (LASHA) protocol remains unclear. This study evaluates the relations...

Full description

Saved in:
Bibliographic Details
Main Authors: Zehra Dhanani, Omar Al Omari, Alonso Marquez, Kartik Shenoy, Fatima Anjum
Format: Article
Language:English
Published: Elsevier 2025-08-01
Series:JHLT Open
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S2950133425001004
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849389486682669056
author Zehra Dhanani
Omar Al Omari
Alonso Marquez
Kartik Shenoy
Fatima Anjum
author_facet Zehra Dhanani
Omar Al Omari
Alonso Marquez
Kartik Shenoy
Fatima Anjum
author_sort Zehra Dhanani
collection DOAJ
description Background: Donor-derived cell-free DNA (dd-cfDNA) is a valuable biomarker for allograft injury, but its association with various histopathologic injury patterns as specified in the Lung Allograft Standardized Histological Analysis (LASHA) protocol remains unclear. This study evaluates the relationship between various histopathologic findings as per the LASHA protocol, dd-cfDNA levels including extreme molecular injury (EMI), and allograft outcomes. Methods: We conducted a retrospective analysis of lung transplant recipients at a single center (2022-2024). A total of 84 patients with 589 dd-cfDNA samples were analyzed, with 124 samples having corresponding transbronchial biopsy (TBBX) and bronchoalveolar lavage (BAL) data. EMI was defined as dd-cfDNA > 5%. Multivariate regression analyses examined associations between histopathologic findings—including hemosiderin-laden macrophages (HLM) and organizing pneumonia (OP)—clinical comorbidities, EMI, and allograft outcomes. Results: EMI was observed in 29% of the cohort and was significantly associated with CLAD (p = 0.04), diabetes (p = 0.009), and elevated DSAs (p = 0.03). EMI was also more frequently detected in Black patients. On histopathology, OP was present in 11.2% of TBBX samples, while 8.9% had HLM. Patients with OP had significantly higher median dd-cfDNA levels compared to those without OP (p < 0.05) and exhibited lymphocytic-predominant BAL (p < 0.001). HLM was more prevalent in Black patients and in those with clinical or radiographic suspicion of OP, even in the absence of OP on histopathology. Conclusion: Our study highlights novel associations between dd-cfDNA, histopathologic findings, comorbidities, race, and allograft injury, emphasizing the need for personalized monitoring and risk stratification in lung transplantation and to explore mechanisms underlying the observed disparities.
format Article
id doaj-art-066daed46e7740bf91446ae1464cd002
institution Kabale University
issn 2950-1334
language English
publishDate 2025-08-01
publisher Elsevier
record_format Article
series JHLT Open
spelling doaj-art-066daed46e7740bf91446ae1464cd0022025-08-20T03:41:57ZengElsevierJHLT Open2950-13342025-08-01910030510.1016/j.jhlto.2025.100305Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipientsZehra Dhanani0Omar Al Omari1Alonso Marquez2Kartik Shenoy3Fatima Anjum4Thoracic Medicine and Surgery, Temple University Hospital, Philadelphia, PA; Corresponding author: Zehra Dhanani, Department of Thoracic Medicine and Surgery, Temple University Hospital, 3401 North Broad Street, Philadelphia, PA 19140.Thoracic Medicine and Surgery, Temple University Hospital, Philadelphia, PAThoracic Medicine and Surgery, Temple University Hospital, Philadelphia, PAThoracic Medicine and Surgery, Lewis Katz School of Medicine at Temple University, Philadelphia, PAThoracic Medicine and Surgery, Lewis Katz School of Medicine at Temple University, Philadelphia, PABackground: Donor-derived cell-free DNA (dd-cfDNA) is a valuable biomarker for allograft injury, but its association with various histopathologic injury patterns as specified in the Lung Allograft Standardized Histological Analysis (LASHA) protocol remains unclear. This study evaluates the relationship between various histopathologic findings as per the LASHA protocol, dd-cfDNA levels including extreme molecular injury (EMI), and allograft outcomes. Methods: We conducted a retrospective analysis of lung transplant recipients at a single center (2022-2024). A total of 84 patients with 589 dd-cfDNA samples were analyzed, with 124 samples having corresponding transbronchial biopsy (TBBX) and bronchoalveolar lavage (BAL) data. EMI was defined as dd-cfDNA > 5%. Multivariate regression analyses examined associations between histopathologic findings—including hemosiderin-laden macrophages (HLM) and organizing pneumonia (OP)—clinical comorbidities, EMI, and allograft outcomes. Results: EMI was observed in 29% of the cohort and was significantly associated with CLAD (p = 0.04), diabetes (p = 0.009), and elevated DSAs (p = 0.03). EMI was also more frequently detected in Black patients. On histopathology, OP was present in 11.2% of TBBX samples, while 8.9% had HLM. Patients with OP had significantly higher median dd-cfDNA levels compared to those without OP (p < 0.05) and exhibited lymphocytic-predominant BAL (p < 0.001). HLM was more prevalent in Black patients and in those with clinical or radiographic suspicion of OP, even in the absence of OP on histopathology. Conclusion: Our study highlights novel associations between dd-cfDNA, histopathologic findings, comorbidities, race, and allograft injury, emphasizing the need for personalized monitoring and risk stratification in lung transplantation and to explore mechanisms underlying the observed disparities.http://www.sciencedirect.com/science/article/pii/S2950133425001004Organizing pneumoniaHemosiderin laden macrophagesAllograft injuryChronic lung allograft dysfunctionImmune-mediated injury
spellingShingle Zehra Dhanani
Omar Al Omari
Alonso Marquez
Kartik Shenoy
Fatima Anjum
Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipients
JHLT Open
Organizing pneumonia
Hemosiderin laden macrophages
Allograft injury
Chronic lung allograft dysfunction
Immune-mediated injury
title Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipients
title_full Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipients
title_fullStr Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipients
title_full_unstemmed Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipients
title_short Unique association of extreme elevation of cell free DNA and histologic patterns of intra-alveolar injury among lung transplant recipients
title_sort unique association of extreme elevation of cell free dna and histologic patterns of intra alveolar injury among lung transplant recipients
topic Organizing pneumonia
Hemosiderin laden macrophages
Allograft injury
Chronic lung allograft dysfunction
Immune-mediated injury
url http://www.sciencedirect.com/science/article/pii/S2950133425001004
work_keys_str_mv AT zehradhanani uniqueassociationofextremeelevationofcellfreednaandhistologicpatternsofintraalveolarinjuryamonglungtransplantrecipients
AT omaralomari uniqueassociationofextremeelevationofcellfreednaandhistologicpatternsofintraalveolarinjuryamonglungtransplantrecipients
AT alonsomarquez uniqueassociationofextremeelevationofcellfreednaandhistologicpatternsofintraalveolarinjuryamonglungtransplantrecipients
AT kartikshenoy uniqueassociationofextremeelevationofcellfreednaandhistologicpatternsofintraalveolarinjuryamonglungtransplantrecipients
AT fatimaanjum uniqueassociationofextremeelevationofcellfreednaandhistologicpatternsofintraalveolarinjuryamonglungtransplantrecipients