Screening Driving Transcription Factors in the Processing of Gastric Cancer

Background. Construction of the transcriptional regulatory network can provide additional clues on the regulatory mechanisms and therapeutic applications in gastric cancer. Methods. Gene expression profiles of gastric cancer were downloaded from GEO database for integrated analysis. All of DEGs were...

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Main Authors: Guangzhong Xu, Kai Li, Nengwei Zhang, Bin Zhu, Guosheng Feng
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:Gastroenterology Research and Practice
Online Access:http://dx.doi.org/10.1155/2016/8431480
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author Guangzhong Xu
Kai Li
Nengwei Zhang
Bin Zhu
Guosheng Feng
author_facet Guangzhong Xu
Kai Li
Nengwei Zhang
Bin Zhu
Guosheng Feng
author_sort Guangzhong Xu
collection DOAJ
description Background. Construction of the transcriptional regulatory network can provide additional clues on the regulatory mechanisms and therapeutic applications in gastric cancer. Methods. Gene expression profiles of gastric cancer were downloaded from GEO database for integrated analysis. All of DEGs were analyzed by GO enrichment and KEGG pathway enrichment. Transcription factors were further identified and then a global transcriptional regulatory network was constructed. Results. By integrated analysis of the six eligible datasets (340 cases and 43 controls), a bunch of 2327 DEGs were identified, including 2100 upregulated and 227 downregulated DEGs. Functional enrichment analysis of DEGs showed that digestion was a significantly enriched GO term for biological process. Moreover, there were two important enriched KEGG pathways: cell cycle and homologous recombination. Furthermore, a total of 70 differentially expressed TFs were identified and the transcriptional regulatory network was constructed, which consisted of 566 TF-target interactions. The top ten TFs regulating most downstream target genes were BRCA1, ARID3A, EHF, SOX10, ZNF263, FOXL1, FEV, GATA3, FOXC1, and FOXD1. Most of them were involved in the carcinogenesis of gastric cancer. Conclusion. The transcriptional regulatory network can help researchers to further clarify the underlying regulatory mechanisms of gastric cancer tumorigenesis.
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spelling doaj-art-02599646dd794dad81674d2bc1a282bc2025-08-20T02:01:49ZengWileyGastroenterology Research and Practice1687-61211687-630X2016-01-01201610.1155/2016/84314808431480Screening Driving Transcription Factors in the Processing of Gastric CancerGuangzhong Xu0Kai Li1Nengwei Zhang2Bin Zhu3Guosheng Feng4Department of General Surgery, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, ChinaDepartment of General Surgery, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, ChinaDepartment of General Surgery, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, ChinaDepartment of General Surgery, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, ChinaDepartment of General Surgery, Beijing Chao-Yang Hospital, Capital Medical University, Beijing 100020, ChinaBackground. Construction of the transcriptional regulatory network can provide additional clues on the regulatory mechanisms and therapeutic applications in gastric cancer. Methods. Gene expression profiles of gastric cancer were downloaded from GEO database for integrated analysis. All of DEGs were analyzed by GO enrichment and KEGG pathway enrichment. Transcription factors were further identified and then a global transcriptional regulatory network was constructed. Results. By integrated analysis of the six eligible datasets (340 cases and 43 controls), a bunch of 2327 DEGs were identified, including 2100 upregulated and 227 downregulated DEGs. Functional enrichment analysis of DEGs showed that digestion was a significantly enriched GO term for biological process. Moreover, there were two important enriched KEGG pathways: cell cycle and homologous recombination. Furthermore, a total of 70 differentially expressed TFs were identified and the transcriptional regulatory network was constructed, which consisted of 566 TF-target interactions. The top ten TFs regulating most downstream target genes were BRCA1, ARID3A, EHF, SOX10, ZNF263, FOXL1, FEV, GATA3, FOXC1, and FOXD1. Most of them were involved in the carcinogenesis of gastric cancer. Conclusion. The transcriptional regulatory network can help researchers to further clarify the underlying regulatory mechanisms of gastric cancer tumorigenesis.http://dx.doi.org/10.1155/2016/8431480
spellingShingle Guangzhong Xu
Kai Li
Nengwei Zhang
Bin Zhu
Guosheng Feng
Screening Driving Transcription Factors in the Processing of Gastric Cancer
Gastroenterology Research and Practice
title Screening Driving Transcription Factors in the Processing of Gastric Cancer
title_full Screening Driving Transcription Factors in the Processing of Gastric Cancer
title_fullStr Screening Driving Transcription Factors in the Processing of Gastric Cancer
title_full_unstemmed Screening Driving Transcription Factors in the Processing of Gastric Cancer
title_short Screening Driving Transcription Factors in the Processing of Gastric Cancer
title_sort screening driving transcription factors in the processing of gastric cancer
url http://dx.doi.org/10.1155/2016/8431480
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