PLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axis

Abstract Background Circular RNA (circRNA) plays a pivotal role in regulating nucleus pulposus cells (NPCs) function, making it a promising therapeutic target for intervertebral disc degeneration (IDD). This study aimed to identify circRNA closely associated with IDD and assess their potential as th...

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Main Authors: Shouliang Xiong, Quanlai Zhao, Yu Zhang, Yifeng Li, Xiaoming Yang, Liang Xiao
Format: Article
Language:English
Published: BMC 2025-07-01
Series:Journal of Translational Medicine
Subjects:
Online Access:https://doi.org/10.1186/s12967-025-06800-z
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author Shouliang Xiong
Quanlai Zhao
Yu Zhang
Yifeng Li
Xiaoming Yang
Liang Xiao
author_facet Shouliang Xiong
Quanlai Zhao
Yu Zhang
Yifeng Li
Xiaoming Yang
Liang Xiao
author_sort Shouliang Xiong
collection DOAJ
description Abstract Background Circular RNA (circRNA) plays a pivotal role in regulating nucleus pulposus cells (NPCs) function, making it a promising therapeutic target for intervertebral disc degeneration (IDD). This study aimed to identify circRNA closely associated with IDD and assess their potential as therapeutic target. Methods Circ_0003251 was identified via circRNA sequencing and validated in degenerated human nucleus pulposus (NP) tissues. Its properties were characterized using Sanger sequencing, oligo(dT) primers, RNase R treatment, and fluorescence in situ hybridization. Functional experiments assessed its role in vitro, while molecular mechanisms were explored through luciferase reporter assays, RNA-binding protein immunoprecipitation, and immunofluorescence. To assess therapeutic potential, circ_0003251 was encapsulated in PLGA (poly(lactic-co-glycolic acid)) microspheres (MS) and evaluated in vitro and in vivo. Results Circ_0003251 expression was significantly downregulated in degenerated NPCs and NP tissues. Its upregulation enhanced NPCs proliferation, extracellular matrix synthesis, and reduced apoptosis. Mechanistically, circ_0003251 acted as a miR-637 sponge, inhibiting AKT1 suppression and alleviating NPCs degeneration. PLGA MS successfully delivered circ_0003251, enhancing its therapeutic effect and delaying IDD in vivo. Conclusion Circ_0003251 was a promising biomarker and therapeutic target for IDD. PLGA MS delivery ensured effective application, offering a novel strategy for IDD treatment.
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institution Kabale University
issn 1479-5876
language English
publishDate 2025-07-01
publisher BMC
record_format Article
series Journal of Translational Medicine
spelling doaj-art-0215570427de4595a9880720aecc28cf2025-08-20T03:42:56ZengBMCJournal of Translational Medicine1479-58762025-07-0123111910.1186/s12967-025-06800-zPLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axisShouliang Xiong0Quanlai Zhao1Yu Zhang2Yifeng Li3Xiaoming Yang4Liang Xiao5Department of Orthopedics, Yijishan Hospital of Wannan Medical CollegeDepartment of Spine Surgery, Yijishan Hospital of Wannan Medical CollegeKey Laboratory of Non-Coding RNA Transformation Research of Anhui Higher Education InstitutionDepartment of Spine Surgery, Yijishan Hospital of Wannan Medical CollegeDepartment of Spine Surgery, Yijishan Hospital of Wannan Medical CollegeDepartment of Spine Surgery, Yijishan Hospital of Wannan Medical CollegeAbstract Background Circular RNA (circRNA) plays a pivotal role in regulating nucleus pulposus cells (NPCs) function, making it a promising therapeutic target for intervertebral disc degeneration (IDD). This study aimed to identify circRNA closely associated with IDD and assess their potential as therapeutic target. Methods Circ_0003251 was identified via circRNA sequencing and validated in degenerated human nucleus pulposus (NP) tissues. Its properties were characterized using Sanger sequencing, oligo(dT) primers, RNase R treatment, and fluorescence in situ hybridization. Functional experiments assessed its role in vitro, while molecular mechanisms were explored through luciferase reporter assays, RNA-binding protein immunoprecipitation, and immunofluorescence. To assess therapeutic potential, circ_0003251 was encapsulated in PLGA (poly(lactic-co-glycolic acid)) microspheres (MS) and evaluated in vitro and in vivo. Results Circ_0003251 expression was significantly downregulated in degenerated NPCs and NP tissues. Its upregulation enhanced NPCs proliferation, extracellular matrix synthesis, and reduced apoptosis. Mechanistically, circ_0003251 acted as a miR-637 sponge, inhibiting AKT1 suppression and alleviating NPCs degeneration. PLGA MS successfully delivered circ_0003251, enhancing its therapeutic effect and delaying IDD in vivo. Conclusion Circ_0003251 was a promising biomarker and therapeutic target for IDD. PLGA MS delivery ensured effective application, offering a novel strategy for IDD treatment.https://doi.org/10.1186/s12967-025-06800-zIntervertebral disc degenerationNucleus pulposus cellsCirc_0003251Microspheres
spellingShingle Shouliang Xiong
Quanlai Zhao
Yu Zhang
Yifeng Li
Xiaoming Yang
Liang Xiao
PLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axis
Journal of Translational Medicine
Intervertebral disc degeneration
Nucleus pulposus cells
Circ_0003251
Microspheres
title PLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axis
title_full PLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axis
title_fullStr PLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axis
title_full_unstemmed PLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axis
title_short PLGA-microspheres carried circ_0003251 alleviate intervertebral disc degeneration via the miR-637/AKT1 axis
title_sort plga microspheres carried circ 0003251 alleviate intervertebral disc degeneration via the mir 637 akt1 axis
topic Intervertebral disc degeneration
Nucleus pulposus cells
Circ_0003251
Microspheres
url https://doi.org/10.1186/s12967-025-06800-z
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