RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogramming

Hexavalent chromium [Cr(VI)], one common environmental contaminant, has long been recognized as a carcinogen associated with lung cancer, but roles and mechanisms of Cr(VI)-induced epigenetic dysregulations in carcinogenesis remain to be investigated. In this study, we identified that RNA m5C methyl...

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Main Authors: Rui-Ke Zhang, Yan Li, Fan-Li Sun, Zhi-Hao Zhou, Yun-Xia Xie, Wen-Jing Liu, Wei Wang, Jian-Ge Qiu, Bing-Hua Jiang, Lin Wang
Format: Article
Language:English
Published: Elsevier 2024-10-01
Series:Environment International
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Online Access:http://www.sciencedirect.com/science/article/pii/S016041202400641X
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author Rui-Ke Zhang
Yan Li
Fan-Li Sun
Zhi-Hao Zhou
Yun-Xia Xie
Wen-Jing Liu
Wei Wang
Jian-Ge Qiu
Bing-Hua Jiang
Lin Wang
author_facet Rui-Ke Zhang
Yan Li
Fan-Li Sun
Zhi-Hao Zhou
Yun-Xia Xie
Wen-Jing Liu
Wei Wang
Jian-Ge Qiu
Bing-Hua Jiang
Lin Wang
author_sort Rui-Ke Zhang
collection DOAJ
description Hexavalent chromium [Cr(VI)], one common environmental contaminant, has long been recognized as a carcinogen associated with lung cancer, but roles and mechanisms of Cr(VI)-induced epigenetic dysregulations in carcinogenesis remain to be investigated. In this study, we identified that RNA m5C methyltransferase NSUN2 was significantly upregulated in Cr(VI)-transformed cells and lung tissues of Cr(VI)-exposed mice. Inhibition of NSUN2 reduced cell proliferation, migration, colony formation and tube formation abilities. We found NSUN2-mediated m5C modification induced metabolic reprogramming and cell cycle by promoting the mRNA stabilities of ME1, GLUT3 and CDK2. In addition, knockdown of NSUN2 attenuated tumorigenesis and angiogenesis in vivo. RNA m5C reader ALYREF was identified to be involved in NSUN2-mediated m5C modification in Cr (VI)-induced carcinogenesis. Further study showed that EP300 induced NSUN2 upregulation through transcriptional activation by inducing histone modification at H3K27ac site for regulating Cr(VI) carcinogenesis. Our findings demonstrated novel role and mechanism of NSUN2 and epigenetic changes by increasing the RNA m5C modification that are important for Cr (VI)-induced carcinogenesis through NSUN2/ALYREF pathway. NSUN2, ALYREF, ME1, GLUT3 or/and CDK2 may be used as potential new biomarkers or/and therapeutic target(s) in the future.
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spelling doaj-art-017ba71f7b6e43db87aee787ad35cf7e2025-08-20T02:09:47ZengElsevierEnvironment International0160-41202024-10-0119210905510.1016/j.envint.2024.109055RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogrammingRui-Ke Zhang0Yan Li1Fan-Li Sun2Zhi-Hao Zhou3Yun-Xia Xie4Wen-Jing Liu5Wei Wang6Jian-Ge Qiu7Bing-Hua Jiang8Lin Wang9Tianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, ChinaTianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, ChinaTianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, ChinaTianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, ChinaTianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, ChinaThe Affiliated Cancer Hospital of Zhengzhou University, Zhengzhou 450000, ChinaTianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, ChinaTianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, ChinaTianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, China; Corresponding authors.Tianjian Laboratory of Advanced Biomedical Sciences, Academy of Medical Science, Zhengzhou University, Zhengzhou 450000, China; Corresponding authors.Hexavalent chromium [Cr(VI)], one common environmental contaminant, has long been recognized as a carcinogen associated with lung cancer, but roles and mechanisms of Cr(VI)-induced epigenetic dysregulations in carcinogenesis remain to be investigated. In this study, we identified that RNA m5C methyltransferase NSUN2 was significantly upregulated in Cr(VI)-transformed cells and lung tissues of Cr(VI)-exposed mice. Inhibition of NSUN2 reduced cell proliferation, migration, colony formation and tube formation abilities. We found NSUN2-mediated m5C modification induced metabolic reprogramming and cell cycle by promoting the mRNA stabilities of ME1, GLUT3 and CDK2. In addition, knockdown of NSUN2 attenuated tumorigenesis and angiogenesis in vivo. RNA m5C reader ALYREF was identified to be involved in NSUN2-mediated m5C modification in Cr (VI)-induced carcinogenesis. Further study showed that EP300 induced NSUN2 upregulation through transcriptional activation by inducing histone modification at H3K27ac site for regulating Cr(VI) carcinogenesis. Our findings demonstrated novel role and mechanism of NSUN2 and epigenetic changes by increasing the RNA m5C modification that are important for Cr (VI)-induced carcinogenesis through NSUN2/ALYREF pathway. NSUN2, ALYREF, ME1, GLUT3 or/and CDK2 may be used as potential new biomarkers or/and therapeutic target(s) in the future.http://www.sciencedirect.com/science/article/pii/S016041202400641XHexavalent chromiumNSUN2Cell malignant transformationMetabolic reprogrammingTumor growth
spellingShingle Rui-Ke Zhang
Yan Li
Fan-Li Sun
Zhi-Hao Zhou
Yun-Xia Xie
Wen-Jing Liu
Wei Wang
Jian-Ge Qiu
Bing-Hua Jiang
Lin Wang
RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogramming
Environment International
Hexavalent chromium
NSUN2
Cell malignant transformation
Metabolic reprogramming
Tumor growth
title RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogramming
title_full RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogramming
title_fullStr RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogramming
title_full_unstemmed RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogramming
title_short RNA methyltransferase NSUN2-mediated m5C methylation promotes Cr(VI)-induced malignant transformation and lung cancer by accelerating metabolism reprogramming
title_sort rna methyltransferase nsun2 mediated m5c methylation promotes cr vi induced malignant transformation and lung cancer by accelerating metabolism reprogramming
topic Hexavalent chromium
NSUN2
Cell malignant transformation
Metabolic reprogramming
Tumor growth
url http://www.sciencedirect.com/science/article/pii/S016041202400641X
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