An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variant

Progressive familial intrahepatic cholestasis type 2 (PFIC2) is a rare pediatric cholestatic liver disease caused by genetic deficiency in the bile salt export pump (BSEP, ABCB11). BSEP is an ATP-binding cassette transporter and the primary regulator of hepatic bile acid efflux. Loss of BSEP functio...

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Main Authors: Eric L. Bell, Jennifer K. Truong, Youhwa Jo, Adrianne Kolpak, Lauren Chunn, Natalie Syverud, Melida Mahinic, Jessica R. Durrant, Eitan Hoch, Bharat Reddy, Patrick Stoiber, John P. Miller, Yong Ren, Jonathan Moore, Robert O. Hughes, Alastair S. Garfield
Format: Article
Language:English
Published: Elsevier 2025-07-01
Series:Journal of Lipid Research
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Online Access:http://www.sciencedirect.com/science/article/pii/S0022227525001002
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author Eric L. Bell
Jennifer K. Truong
Youhwa Jo
Adrianne Kolpak
Lauren Chunn
Natalie Syverud
Melida Mahinic
Jessica R. Durrant
Eitan Hoch
Bharat Reddy
Patrick Stoiber
John P. Miller
Yong Ren
Jonathan Moore
Robert O. Hughes
Alastair S. Garfield
author_facet Eric L. Bell
Jennifer K. Truong
Youhwa Jo
Adrianne Kolpak
Lauren Chunn
Natalie Syverud
Melida Mahinic
Jessica R. Durrant
Eitan Hoch
Bharat Reddy
Patrick Stoiber
John P. Miller
Yong Ren
Jonathan Moore
Robert O. Hughes
Alastair S. Garfield
author_sort Eric L. Bell
collection DOAJ
description Progressive familial intrahepatic cholestasis type 2 (PFIC2) is a rare pediatric cholestatic liver disease caused by genetic deficiency in the bile salt export pump (BSEP, ABCB11). BSEP is an ATP-binding cassette transporter and the primary regulator of hepatic bile acid efflux. Loss of BSEP function in PFIC2 leads to cholestasis and intrahepatic accumulation of bile acids, the native toxicity of which drives progressive liver injury, in a manner that correlates with ABCB11 genotype. Here, to support ongoing PFIC2 research, we present two novel translational tools, 1) a codified evidence-based catalog of published disease relevant ABCB11 mutations and 2) a knockin mouse model of the PFIC2-associated missense variant E297G. Using a combination of AI-based indexing of the literature and manual review, we identified 476 nonbenign ABCB11 variants in published patients with cholestatic disease, of which 240 were associated with PFIC2. Additionally, we present phenotypic validation of a novel knockin mouse model of the cholestasis-associated ABCB11 E297G variant. BsepE297G homozygous mice recapitulate the core molecular and pathophysiological aspects of PFIC2, including perturbed Bsep processing and membrane trafficking, cholestasis, and hepatotoxicity. Moreover, and consistent with clinical data, pharmacological ileal bile acid transporter inhibition improved the cholestatic phenotype of BsepE297G mice through increased fecal bile acid excretion. Together, these tools can support clinical and translational efforts to advance understanding and treatment of PFIC2.
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spelling doaj-art-ca30c4099bc04cb89ffbe40c6ca1a17d2025-08-20T03:14:34ZengElsevierJournal of Lipid Research0022-22752025-07-0166710084010.1016/j.jlr.2025.100840An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variantEric L. Bell0Jennifer K. Truong1Youhwa Jo2Adrianne Kolpak3Lauren Chunn4Natalie Syverud5Melida Mahinic6Jessica R. Durrant7Eitan Hoch8Bharat Reddy9Patrick Stoiber10John P. Miller11Yong Ren12Jonathan Moore13Robert O. Hughes14Alastair S. Garfield15Rectify Pharmaceuticals, Cambridge, MA, USA; For correspondence: Eric L. Bell; Robert O. HughesRectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USAGenomenon, Ann Arbor, MI, USAGenomenon, Ann Arbor, MI, USAGenomenon, Ann Arbor, MI, USADTR Labs, Farmers Branch, TX, USARectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USARectify Pharmaceuticals, Cambridge, MA, USA; For correspondence: Eric L. Bell; Robert O. HughesRectify Pharmaceuticals, Cambridge, MA, USAProgressive familial intrahepatic cholestasis type 2 (PFIC2) is a rare pediatric cholestatic liver disease caused by genetic deficiency in the bile salt export pump (BSEP, ABCB11). BSEP is an ATP-binding cassette transporter and the primary regulator of hepatic bile acid efflux. Loss of BSEP function in PFIC2 leads to cholestasis and intrahepatic accumulation of bile acids, the native toxicity of which drives progressive liver injury, in a manner that correlates with ABCB11 genotype. Here, to support ongoing PFIC2 research, we present two novel translational tools, 1) a codified evidence-based catalog of published disease relevant ABCB11 mutations and 2) a knockin mouse model of the PFIC2-associated missense variant E297G. Using a combination of AI-based indexing of the literature and manual review, we identified 476 nonbenign ABCB11 variants in published patients with cholestatic disease, of which 240 were associated with PFIC2. Additionally, we present phenotypic validation of a novel knockin mouse model of the cholestasis-associated ABCB11 E297G variant. BsepE297G homozygous mice recapitulate the core molecular and pathophysiological aspects of PFIC2, including perturbed Bsep processing and membrane trafficking, cholestasis, and hepatotoxicity. Moreover, and consistent with clinical data, pharmacological ileal bile acid transporter inhibition improved the cholestatic phenotype of BsepE297G mice through increased fecal bile acid excretion. Together, these tools can support clinical and translational efforts to advance understanding and treatment of PFIC2.http://www.sciencedirect.com/science/article/pii/S0022227525001002PFIC2Abcb11BSEPcholestasisbile acidmissense
spellingShingle Eric L. Bell
Jennifer K. Truong
Youhwa Jo
Adrianne Kolpak
Lauren Chunn
Natalie Syverud
Melida Mahinic
Jessica R. Durrant
Eitan Hoch
Bharat Reddy
Patrick Stoiber
John P. Miller
Yong Ren
Jonathan Moore
Robert O. Hughes
Alastair S. Garfield
An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variant
Journal of Lipid Research
PFIC2
Abcb11
BSEP
cholestasis
bile acid
missense
title An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variant
title_full An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variant
title_fullStr An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variant
title_full_unstemmed An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variant
title_short An ABCB11 variant registry and novel knockin mouse model of PFIC2 based on the clinically relevant ABCB11 E297G variant
title_sort abcb11 variant registry and novel knockin mouse model of pfic2 based on the clinically relevant abcb11 e297g variant
topic PFIC2
Abcb11
BSEP
cholestasis
bile acid
missense
url http://www.sciencedirect.com/science/article/pii/S0022227525001002
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