COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysis

Background: While epidemiological data suggest a connection between atopic dermatitis (AD) and COVID-19, the molecular mechanisms underlying this relationship remain unclear. Objective: To investigate whether COVID-19-related CpGs may contribute to AD development and whether this association is medi...

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Main Authors: Zhenwei Tang, MD, Yu Chen, MBBS, Yuzhen Ouyang, MBBS, Yu Peng, MD, PhD, Xiaoyong Man, MD, PhD
Format: Article
Language:English
Published: Elsevier 2025-01-01
Series:World Allergy Organization Journal
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Online Access:http://www.sciencedirect.com/science/article/pii/S1939455124001546
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author Zhenwei Tang, MD
Yu Chen, MBBS
Yuzhen Ouyang, MBBS
Yu Peng, MD, PhD
Xiaoyong Man, MD, PhD
author_facet Zhenwei Tang, MD
Yu Chen, MBBS
Yuzhen Ouyang, MBBS
Yu Peng, MD, PhD
Xiaoyong Man, MD, PhD
author_sort Zhenwei Tang, MD
collection DOAJ
description Background: While epidemiological data suggest a connection between atopic dermatitis (AD) and COVID-19, the molecular mechanisms underlying this relationship remain unclear. Objective: To investigate whether COVID-19-related CpGs may contribute to AD development and whether this association is mediated through the regulation of specific genes’ expression. Methods: We combined Mendelian randomization and transcriptome analysis for data-driven explorations. Results: Among the 172 CpGs -associated with COVID-19 infection, merely 3 of them exhibited significant impacts on the risk of AD, including cg04543273, cg11916609, and cg10636246. In the following analysis of the causal effects of CpGs and their related gene expression, cg04543273 inhibited LMAN2 expression. However, there was not a significant impact of cg11916609 and cg10636246 on the expression of their corresponding genes. Besides, transcriptome analysis suggested that LMAN2 expression was significantly upregulated among the COVID-19-infected population, and LMAN2 expression was obviously correlated with Type 2 helper cells across different post-infection time points. Conclusion: Overall, this study provides new insights of the COVID-19-related onset and exacerbation of AD-COVID-19-related epigenetic changes and their regulatory impact on transcription. A novel role of LMAN2 was proposed in the relationship between viral infection and AD. More studies are warranted to further explore the mechanism of LMAN2-related immunopathology.
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spelling doaj-art-84bae295cde240a78f4385dcf55e63902025-01-17T04:49:17ZengElsevierWorld Allergy Organization Journal1939-45512025-01-01181101022COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysisZhenwei Tang, MD0Yu Chen, MBBS1Yuzhen Ouyang, MBBS2Yu Peng, MD, PhD3Xiaoyong Man, MD, PhD4Department of Dermatology, Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China; Corresponding author. 88 Jiefang Road, Hangzhou, Zhejiang, China.Clinical Medicine Eight-year Program, Xiangya Hospital, Central South University, Changsha, ChinaClinical Medicine Eight-year Program, Xiangya Hospital, Central South University, Changsha, ChinaDepartment of Rheumatology, Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, ChinaDepartment of Dermatology, Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China; Corresponding author.Background: While epidemiological data suggest a connection between atopic dermatitis (AD) and COVID-19, the molecular mechanisms underlying this relationship remain unclear. Objective: To investigate whether COVID-19-related CpGs may contribute to AD development and whether this association is mediated through the regulation of specific genes’ expression. Methods: We combined Mendelian randomization and transcriptome analysis for data-driven explorations. Results: Among the 172 CpGs -associated with COVID-19 infection, merely 3 of them exhibited significant impacts on the risk of AD, including cg04543273, cg11916609, and cg10636246. In the following analysis of the causal effects of CpGs and their related gene expression, cg04543273 inhibited LMAN2 expression. However, there was not a significant impact of cg11916609 and cg10636246 on the expression of their corresponding genes. Besides, transcriptome analysis suggested that LMAN2 expression was significantly upregulated among the COVID-19-infected population, and LMAN2 expression was obviously correlated with Type 2 helper cells across different post-infection time points. Conclusion: Overall, this study provides new insights of the COVID-19-related onset and exacerbation of AD-COVID-19-related epigenetic changes and their regulatory impact on transcription. A novel role of LMAN2 was proposed in the relationship between viral infection and AD. More studies are warranted to further explore the mechanism of LMAN2-related immunopathology.http://www.sciencedirect.com/science/article/pii/S1939455124001546COVID-19Atopic dermatitisDNA methylationsLMAN2 proteinEpigenetics
spellingShingle Zhenwei Tang, MD
Yu Chen, MBBS
Yuzhen Ouyang, MBBS
Yu Peng, MD, PhD
Xiaoyong Man, MD, PhD
COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysis
World Allergy Organization Journal
COVID-19
Atopic dermatitis
DNA methylations
LMAN2 protein
Epigenetics
title COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysis
title_full COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysis
title_fullStr COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysis
title_full_unstemmed COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysis
title_short COVID-19 related epigenetic changes and atopic dermatitis: An exploratory analysis
title_sort covid 19 related epigenetic changes and atopic dermatitis an exploratory analysis
topic COVID-19
Atopic dermatitis
DNA methylations
LMAN2 protein
Epigenetics
url http://www.sciencedirect.com/science/article/pii/S1939455124001546
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