Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA Pathway

Perineural invasion (PNI) is an important factor leading to the recurrence of pancreatic cancer (PanCa). The NGF–TrkA pathway is related to PNI progression. Ganoderma spore lipid (GSL) is a drug with anti-cancer properties. In this study, we find out whether GSL can prevent PNI of PanCa by inhibitin...

Full description

Saved in:
Bibliographic Details
Main Authors: Haohui Lin MS, Manhon Chung MD, Yi Yang MS, Li Zhang MS, Xiaohua Pan MD, Sa Cai MD, PhD, Yu Pan PhD
Format: Article
Language:English
Published: SAGE Publishing 2025-07-01
Series:Journal of Evidence-Based Integrative Medicine
Online Access:https://doi.org/10.1177/2515690X251356473
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849416000378765312
author Haohui Lin MS
Manhon Chung MD
Yi Yang MS
Li Zhang MS
Xiaohua Pan MD
Sa Cai MD, PhD
Yu Pan PhD
author_facet Haohui Lin MS
Manhon Chung MD
Yi Yang MS
Li Zhang MS
Xiaohua Pan MD
Sa Cai MD, PhD
Yu Pan PhD
author_sort Haohui Lin MS
collection DOAJ
description Perineural invasion (PNI) is an important factor leading to the recurrence of pancreatic cancer (PanCa). The NGF–TrkA pathway is related to PNI progression. Ganoderma spore lipid (GSL) is a drug with anti-cancer properties. In this study, we find out whether GSL can prevent PNI of PanCa by inhibiting NGF–TrkA pathway. In vitro , wound healing assays, transwell-based assays and three-dimensional tumor-nerve cell co-culture system showed that GSL significantly inhibited the migration and invasion capacity of PanCa cells. Inhibiting the NGF–TrkA pathway is considered an effective approach for treating PanCa. We showed that GSL effectively inhibited NGF-TrkA pathway via immunofluorescence assays and western blotting analysis. The supplement of recombinant NGF reversed the GSL inhibitory effect on the migration and invasion of PANC-1. In vivo , a sciatic nerve invasion animal model was constructed using BALB/c mice. GSL significantly suppressed tumor growth and suppressed the expression of TrkA, NGF, and vimentin and upregulated the level of the epithelial marker E-Cadherin. Moreover, GSL reduced the expression of S100 and PGP9.5. These findings suggested that GSL effectively inhibited the PNI of PanCa cells by downregulating the NGF–TrkA pathway, which may provide a new adjuvant for PanCa treatment.
format Article
id doaj-art-7f89107c4c5a47bdbfdcf908dece60fb
institution Kabale University
issn 2515-690X
language English
publishDate 2025-07-01
publisher SAGE Publishing
record_format Article
series Journal of Evidence-Based Integrative Medicine
spelling doaj-art-7f89107c4c5a47bdbfdcf908dece60fb2025-08-20T03:33:18ZengSAGE PublishingJournal of Evidence-Based Integrative Medicine2515-690X2025-07-013010.1177/2515690X251356473Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA PathwayHaohui Lin MS0Manhon Chung MD1Yi Yang MS2Li Zhang MS3Xiaohua Pan MD4Sa Cai MD, PhD5Yu Pan PhD6 Laboratory of Regenerative Medicine, The 2nd Affiliated Hospital of Shenzhen University, Medical School, , Shenzhen, China Department of Plastic and Reconstructive Surgery, Shanghai Ninth People's Hospital, , Shanghai, China Laboratory of Regenerative Medicine, The 2nd Affiliated Hospital of Shenzhen University, Medical School, , Shenzhen, China Laboratory of Regenerative Medicine, The 2nd Affiliated Hospital of Shenzhen University, Medical School, , Shenzhen, China Laboratory of Regenerative Medicine, The 2nd Affiliated Hospital of Shenzhen University, Medical School, , Shenzhen, China Laboratory of Regenerative Medicine, The 2nd Affiliated Hospital of Shenzhen University, Medical School, , Shenzhen, China Laboratory of Regenerative Medicine, The 2nd Affiliated Hospital of Shenzhen University, Medical School, , Shenzhen, ChinaPerineural invasion (PNI) is an important factor leading to the recurrence of pancreatic cancer (PanCa). The NGF–TrkA pathway is related to PNI progression. Ganoderma spore lipid (GSL) is a drug with anti-cancer properties. In this study, we find out whether GSL can prevent PNI of PanCa by inhibiting NGF–TrkA pathway. In vitro , wound healing assays, transwell-based assays and three-dimensional tumor-nerve cell co-culture system showed that GSL significantly inhibited the migration and invasion capacity of PanCa cells. Inhibiting the NGF–TrkA pathway is considered an effective approach for treating PanCa. We showed that GSL effectively inhibited NGF-TrkA pathway via immunofluorescence assays and western blotting analysis. The supplement of recombinant NGF reversed the GSL inhibitory effect on the migration and invasion of PANC-1. In vivo , a sciatic nerve invasion animal model was constructed using BALB/c mice. GSL significantly suppressed tumor growth and suppressed the expression of TrkA, NGF, and vimentin and upregulated the level of the epithelial marker E-Cadherin. Moreover, GSL reduced the expression of S100 and PGP9.5. These findings suggested that GSL effectively inhibited the PNI of PanCa cells by downregulating the NGF–TrkA pathway, which may provide a new adjuvant for PanCa treatment.https://doi.org/10.1177/2515690X251356473
spellingShingle Haohui Lin MS
Manhon Chung MD
Yi Yang MS
Li Zhang MS
Xiaohua Pan MD
Sa Cai MD, PhD
Yu Pan PhD
Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA Pathway
Journal of Evidence-Based Integrative Medicine
title Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA Pathway
title_full Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA Pathway
title_fullStr Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA Pathway
title_full_unstemmed Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA Pathway
title_short Ganoderma Spore Lipids Inhibit Perineural Invasion in Pancreatic Cancer by Downregulating NGF-TrkA Pathway
title_sort ganoderma spore lipids inhibit perineural invasion in pancreatic cancer by downregulating ngf trka pathway
url https://doi.org/10.1177/2515690X251356473
work_keys_str_mv AT haohuilinms ganodermasporelipidsinhibitperineuralinvasioninpancreaticcancerbydownregulatingngftrkapathway
AT manhonchungmd ganodermasporelipidsinhibitperineuralinvasioninpancreaticcancerbydownregulatingngftrkapathway
AT yiyangms ganodermasporelipidsinhibitperineuralinvasioninpancreaticcancerbydownregulatingngftrkapathway
AT lizhangms ganodermasporelipidsinhibitperineuralinvasioninpancreaticcancerbydownregulatingngftrkapathway
AT xiaohuapanmd ganodermasporelipidsinhibitperineuralinvasioninpancreaticcancerbydownregulatingngftrkapathway
AT sacaimdphd ganodermasporelipidsinhibitperineuralinvasioninpancreaticcancerbydownregulatingngftrkapathway
AT yupanphd ganodermasporelipidsinhibitperineuralinvasioninpancreaticcancerbydownregulatingngftrkapathway