Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasis

Mitochondrial intermembrane space (IMS) harbors a series of small, evolutionarily conserved redox-active cysteine-rich proteins. These proteins are essential for the functioning of cytochrome c oxidase, but their role in maintaining cellular redox processes is unknown. Here, we find out that in the...

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Main Authors: Soumyajit Mukherjee, Shubhojit Das, Sourav Kumar Patra, Mayukh Das, Sanjay Ghosh, Alok Ghosh
Format: Article
Language:English
Published: Elsevier 2024-12-01
Series:Advances in Redox Research
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Online Access:http://www.sciencedirect.com/science/article/pii/S2667137924000195
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author Soumyajit Mukherjee
Shubhojit Das
Sourav Kumar Patra
Mayukh Das
Sanjay Ghosh
Alok Ghosh
author_facet Soumyajit Mukherjee
Shubhojit Das
Sourav Kumar Patra
Mayukh Das
Sanjay Ghosh
Alok Ghosh
author_sort Soumyajit Mukherjee
collection DOAJ
description Mitochondrial intermembrane space (IMS) harbors a series of small, evolutionarily conserved redox-active cysteine-rich proteins. These proteins are essential for the functioning of cytochrome c oxidase, but their role in maintaining cellular redox processes is unknown. Here, we find out that in the absence of two such cysteine-rich Cx9C-Cx10C proteins, cytochrome c oxidase subunit 12 (Cox12) or cytochrome c oxidase assembly factor 6 (Coa6), Saccharomyces cerevisiae cells become sensitive under the oxidative and nitrosative stress. Interestingly, knockout of COX12 generates a significant amount of endogenous reactive oxygen species (ROS) and reactive nitrogen species (RNS) as evidenced by FACS analysis. Moreover, cellular redox status, redox-active enzymes glutathione reductase, catalase, S-nitroso glutathione reductase, and protein nitration were significantly affected in Cox12 null cells. Further, we found that an overexpression of COX12 partially protects mitochondrial respiratory subunit Sdh2 under oxidative and nitrosative stress. Taken together, we provide proof of evidence that cysteine-rich proteins in the IMS dynamically control the cellular redox milieu and actively prevent reactive nitrogen and oxygen species generation.
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spelling doaj-art-798064ff94d142ccb2dc4583c4c0b9582024-12-12T05:24:00ZengElsevierAdvances in Redox Research2667-13792024-12-0113100112Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasisSoumyajit Mukherjee0Shubhojit Das1Sourav Kumar Patra2Mayukh Das3Sanjay Ghosh4Alok Ghosh5Department of Biochemistry, University of Calcutta, 35 Ballygunge Circular Road, Kolkata 700019, IndiaDepartment of Biochemistry, University of Calcutta, 35 Ballygunge Circular Road, Kolkata 700019, India; Department of Biochemistry, Ramkrishna Ashram Diagnostic and Research Institute, Nimpith 743338, IndiaDepartment of Biochemistry, University of Calcutta, 35 Ballygunge Circular Road, Kolkata 700019, India; Post Doctoral Research Associate, Dept. of Biochemistry and Biophysics, University of North Carolina, Chapel Hill, NC, USADepartment of Biochemistry, University of Calcutta, 35 Ballygunge Circular Road, Kolkata 700019, IndiaDepartment of Biochemistry, University of Calcutta, 35 Ballygunge Circular Road, Kolkata 700019, IndiaDepartment of Biochemistry, University of Calcutta, 35 Ballygunge Circular Road, Kolkata 700019, India; Corresponding author at: Department of Biochemistry, University of Calcutta, 35 Ballygunge Circular Road, Kolkata 700019, India.Mitochondrial intermembrane space (IMS) harbors a series of small, evolutionarily conserved redox-active cysteine-rich proteins. These proteins are essential for the functioning of cytochrome c oxidase, but their role in maintaining cellular redox processes is unknown. Here, we find out that in the absence of two such cysteine-rich Cx9C-Cx10C proteins, cytochrome c oxidase subunit 12 (Cox12) or cytochrome c oxidase assembly factor 6 (Coa6), Saccharomyces cerevisiae cells become sensitive under the oxidative and nitrosative stress. Interestingly, knockout of COX12 generates a significant amount of endogenous reactive oxygen species (ROS) and reactive nitrogen species (RNS) as evidenced by FACS analysis. Moreover, cellular redox status, redox-active enzymes glutathione reductase, catalase, S-nitroso glutathione reductase, and protein nitration were significantly affected in Cox12 null cells. Further, we found that an overexpression of COX12 partially protects mitochondrial respiratory subunit Sdh2 under oxidative and nitrosative stress. Taken together, we provide proof of evidence that cysteine-rich proteins in the IMS dynamically control the cellular redox milieu and actively prevent reactive nitrogen and oxygen species generation.http://www.sciencedirect.com/science/article/pii/S2667137924000195Cytochrome c oxidase subunit 12 (Cox12)Mitochondrial respirationCellular redox statusOxidative and nitrosative stresses
spellingShingle Soumyajit Mukherjee
Shubhojit Das
Sourav Kumar Patra
Mayukh Das
Sanjay Ghosh
Alok Ghosh
Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasis
Advances in Redox Research
Cytochrome c oxidase subunit 12 (Cox12)
Mitochondrial respiration
Cellular redox status
Oxidative and nitrosative stresses
title Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasis
title_full Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasis
title_fullStr Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasis
title_full_unstemmed Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasis
title_short Absence of mitochondrial CX9C-CX10C protein Cox12 generates oxidative and nitrosative stress in Saccharomyces cerevisiae: Implication on cellular redox homeostasis
title_sort absence of mitochondrial cx9c cx10c protein cox12 generates oxidative and nitrosative stress in saccharomyces cerevisiae implication on cellular redox homeostasis
topic Cytochrome c oxidase subunit 12 (Cox12)
Mitochondrial respiration
Cellular redox status
Oxidative and nitrosative stresses
url http://www.sciencedirect.com/science/article/pii/S2667137924000195
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